D. H. Limoli

Pseudomonas aeruginosa surface motility and invasion into competing communities enhances interspecies antagonism

This study shows that Pseudomonas aeruginosa uses type IV pili-mediated chemotaxis to invade and disrupt Staphylococcus aureus microcolonies, enhancing interspecies antagonism.

The great divide: rhamnolipids mediate separation between P. aeruginosa and S. aureus

This study shows that rhamnolipids mediate physical separation between P. aeruginosa and S. aureus, enabling coexistence through surfactant flow deflection.

Staphylococcal secreted cytotoxins are competition sensing signals for Pseudomonas aeruginosa

This study shows Pseudomonas aeruginosa uses Staphylococcus aureus secreted toxins as signals to detect competition and activate its type six secretion system to outcompete S. aureus during coinfection.

Twitching cells use a chemoreceptor to detect bacterial competitors

This study identifies PilJ as a chemoreceptor in Pseudomonas aeruginosa that detects bacterial competitors, enabling directional motility through methylation and demethylation by PilK and ChpB.

Interspecies signaling generates exploratory motility in Pseudomonas aeruginosa

This study shows Pseudomonas aeruginosa changes its motility behavior in response to signals from Staphylococcus aureus, leading to directed movement and colony invasion.