Author

D. M. Bautista

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biophysics· 1 Evolutionary Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:cryo-EM· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

R. B. Brem· 2 shared A. Kotecha· 1 shared B. Sorum· 1 shared C. M. Hoel· 1 shared C. M. Walsh· 1 shared D. B. Toso· 1 shared D. M. Kern· 1 shared E. C. Brock· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersEvolutionary Biology

R. B. Brem

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Stem cell transcriptional profiles from mouse subspecies reveal cis-regulatory evolution at translation genes

N. M. Simon, Y. Kim, D. M. Bautista, J. R. Dutton, R. B. Brem

The paper identifies cis-regulatory evolution in translation genes in stem cells of M. m. castaneus compared to sister taxa using expression and population-genomic data.

bioRxiv · Jul 18, 2023 · PreprintEvolutionary Biology

Cryo-EM structure of the SARS-CoV-2 3a ion channel in lipid nanodiscs

D. M. Kern, B. Sorum, S. S. Mali, C. M. Hoel, S. Sridharan, J. P. Remis, D. B. Toso, A. Kotecha, D. M. Bautista, S. G. Brohawn

This study presents the cryo-EM structure of SARS-CoV-2 3a ion channel in lipid nanodiscs, revealing its functional properties and evolutionary conservation.

Nature Structural & Molecular Biology · Jun 22, 2021Peer-reviewedBiophysics

Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis

C. M. Walsh, R. Z. Hill, J. Schwendinger-Schreck, J. Deguine, E. C. Brock, N. Kucirek, Z. Rifi, J. Wei, K. Gronert, R. B. Brem, G. M. Barton, D. M. Bautista

This study identifies neutrophils as key initiators of itch in atopic dermatitis and shows that CXCR3 antagonism reduces chronic itch.

bioRxiv · Sep 24, 2019Immunology