Author

D. R. Ingram

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Organism:mouse· 1

Frequent Co-Authors

K. Rai· 3 shared W.-L. Wang· 3 shared A. J. Lazar· 2 shared B. A. Menegaz· 2 shared H. C. Beird· 2 shared J. A. Ludwig· 2 shared M. Maitituoheti· 2 shared S.-E. Lamhamedi-Cherradi· 2 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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3 shared papersCancer Biology

K. Rai

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Author profile →

3 papers

Recent·Most cited

The Androgen Receptor: A Therapeutic Target in Desmoplastic Small Round Cell Sarcoma

S.-E. Lamhamedi-Cherradi, M. Maitituoheti, B. A. Menegaz, S. Krishnan, A. M. Vetter, P. Camacho, C.-C. Wu, H. C. Beird, R. W. Porter, D. R. Ingram, V. Ramamoorthy, S. Mohiuddin, D. McCall, D. D. Truong, B. Cuglievan, , J. A. Ludwig

The paper shows that DSRCT is driven by the androgen receptor and responds to androgen deprivation therapy.

bioRxiv · Jan 20, 2022Cancer Biology

New cellular models of undifferentiated pleomorphic sarcoma and malignant peripheral nerve sheath tumor

A. D. Bhalla, S. M. Landers, A. K. Singh, M. G. Yeagley, G. S. B. Meyerson, Z. A. Mulder, C. B. Delgado-Baez, S. Dunnand, V. Kochat, K. J. Tomczak, T. Nguyen, X. Ma, S. Bolshakov, B. A. Menegaz, S.-E. Lamhamedi-Cherradi, , K. E. Torres

This study generates new cell lines for undifferentiated pleomorphic sarcoma and malignant peripheral nerve sheath tumor to better study sarcomagenesis and develop therapies.

bioRxiv · May 13, 2021 · PreprintCancer Biology

Enhancer Reprogramming Confers Dependence on Glycolysis and IGF signaling in KMT2D Mutant Melanoma

M. Maitituoheti, E. Keung, M. Tang, L. Yan, H. Alam, G. Han, A. Raman, C. Terranova, S. Sarkar, E. Orouji, S. Amin, S. Sharma, M. Williams, N. S. Samant, M. Dhamdhere, , K. Rai

This study identifies KMT2D as a tumor suppressor in melanoma, showing its loss leads to metabolic reprogramming toward glycolysis and dependence on IGF signaling, suggesting therapeutic targeting of these pathways.

bioRxiv · Dec 27, 2018Cancer Biology