Author

D. Shinde

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Approach:genetic perturbation / CRISPR· 2 Research Focus:metabolism & bioenergetics· 2

Frequent Co-Authors

A. Ghosh· 2 shared E. Bueno· 2 shared F. Cava· 2 shared P. Raghu· 2 shared S. Mathre· 2 shared S. Sharma· 2 shared A. Mateus· 1 shared A. Nolan· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

A. Ghosh

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Metabolic reprogramming and flux to cell envelope precursors in a pentose phosphate pathway mutant increases MRSA resistance to β-lactam antibiotics

M. S. Zeden, L. A. Gallagher, E. Bueno, A. Nolan, J. Ahm, D. Shinde, F. Razvi, M. Sladek, O. Burke, E. O'Neill, P. D. Fey, F. Cava, V. C. Thomas, J. P. O'Gara

This study shows that mutating the pentose phosphate pathway gene pgl in MRSA increases resistance to beta-lactam antibiotics through metabolic reprogramming and changes in cell envelope precursors.

bioRxiv · Mar 3, 2023 · PreprintMicrobiology

Transient glycolytic complexation of arsenate enhances resistance in the enteropathogen Vibrio cholerae

E. Bueno, A. Typas, M. Savitski, A. Mateus, V. Pinedo, D. Shinde, V. Thomas, F. Cava

This study identifies a novel arsenate resistance mechanism in V. cholerae involving the formation of a transient glycolytic complex to sequester arsenate and prevent its toxic effects.

bioRxiv · Aug 4, 2022Microbiology

PI3P dependent regulation of cell size and autophagy by phosphatidylinositol 5-phosphate 4-kinase

A. Ghosh, A. Venugopal, D. Shinde, S. M. Sharma, M. Krishnan, S. Mathre, H. Krishnan, R. PADINJAT

This study shows that PIP4K regulates cell size and autophagy in Drosophila by controlling PI3P levels, with implications for neurodegeneration and tumour growth.

bioRxiv · Jun 18, 2022Cell Biology

A novel mass assay to measure phosphatidylinositol-5-phosphate from cells and tissues

A. Ghosh, S. Sharma, D. Shinde, V. Ramya, P. Raghu

This paper introduces a non-radioactive mass assay to measure phosphatidylinositol-5-phosphate (PI5P) levels in Drosophila cells and tissues with high sensitivity.

bioRxiv · Jul 18, 2019Biochemistry

Phosphatidylinositol 5 phosphate 4-kinase regulates insulin sensitivity and PIP3 turnover in Drosophila.

S. Sharma, S. Mathre, R. Visvanathan, D. Shinde, P. Raghu

This study shows that PIP4K regulates PIP3 levels and insulin sensitivity in Drosophila, affecting metabolism and development.

bioRxiv · Jun 25, 2018 · PreprintCell Biology