Author

D. W. Lazinski

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 5

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Organism:Arabidopsis· 2 Organism:bacteria (other)· 1 Approach:biophysical modeling· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Camilli· 3 shared E. Geisinger· 2 shared J. Anthony· 2 shared N. J. Mortman· 2 shared R. R. Isberg· 2 shared S. Syal· 2 shared S. Wood· 2 shared A. Farinha· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersMicrobiology

A. Camilli

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

A genetic interaction screen in Streptococcus pneumoniae identifies functionally redundant vaccine candidate proteins CbpC and CbpJ

L. M. Russo, A. J. Matthews, r. masilamani, D. W. Lazinski, A. Camilli

The paper identifies functionally redundant surface proteins CbpC and CbpJ in S. pneumoniae as potential bivalent vaccine candidates through a genetic interaction screen.

bioRxiv · Apr 7, 2021 · PreprintMicrobiology

A Tail Fiber Protein and a Receptor-Binding Protein Mediate ICP2 Bacteriophage Interactions with Vibrio cholerae OmpU

A. N. W. Lim, M. Yen, K. Seed, D. W. Lazinski, A. Camilli

This study identifies ICP2 phage proteins involved in binding to V. cholerae OmpU and shows that mutations in gp25 and gp23 are required for host-range adaptation.

bioRxiv · Mar 8, 2021Microbiology

Identification of spacer and protospacer sequence requirements in the Vibrio cholerae Type I-E CRISPR/Cas System

J. Bourgeois, D. W. Lazinski, A. Camilli

This study identifies sequence requirements for CRISPR/Cas immunity in Vibrio cholerae, showing that a 5-RTT-3 PAM is necessary for spacer acquisition and interference.

bioRxiv · Aug 9, 2020Microbiology

Antibiotic hypersensitivity signatures identify targets for attack in the Acinetobacter baumannii cell envelope

E. Geisinger, N. J. Mortman, Y. Dai, M. Cokol, S. Syal, A. Farinha, D. Fisher, A. Tang, D. W. Lazinski, S. Wood, J. Anthony, T. Opijnen, R. R. Isberg

This study identifies antibiotic hypersensitivity signatures in A. baumannii to discover new targets for attacking the pathogen's cell envelope.

bioRxiv · Mar 11, 2020Microbiology

The landscape of intrinsic and evolved fluoroquinolone resistance in Acinetobacter baumannii includes suppression of drug-induced prophage replication

E. Geisinger, G. Vargas-Cuebas, N. J. Mortman, S. Syal, E. Wainwright, D. W. Lazinski, S. Wood, Z. Zhu, J. Anthony, T. van Opijnen, R. R. Isberg

This paper identifies mutations that alter fluoroquinolone sensitivity in Acinetobacter baumannii and shows that preferential Topo IV poisoning leads to prophage induction and SOS response activation.

bioRxiv · Oct 14, 2018 · PreprintMicrobiology