Author

Dimitrios Prymidis

German Cancer Research Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Assay:metabolomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:software / tool· 1

Frequent Co-Authors

Christian FrezzaiD· 2 shared Albert Mao LiiD· 1 shared Alexandros P. DrainasiD· 1 shared Ali Taleb AbdallahiD· 1 shared Àlvaro Quintanal-VillalongaiD· 1 shared Andrea C. ChaikovskyiD· 1 shared Anna Edenhofer· 1 shared Bindesh ShresthaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersSystems Biology

Christian FrezzaORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Integrated metabolomics data analysis to generate mechanistic hypotheses with MetaProViz

Christina Schmidt, Jannik Franken, Dénes Türei, Dimitrios Prymidis, Macabe Daley, Christian Frezza, Julio Sáez-Rodríguez

The paper introduces MetaProViz, an R package for metabolomics data analysis that integrates prior knowledge to generate mechanistic hypotheses, using kidney cancer data to demonstrate its capabilities.

Molecular Systems Biology · Aug 17, 2026Peer-reviewedSystems Biology

Cell state-specific metabolic networks govern ferroptosis versus apoptosis in small cell lung cancer

Jun W. Kim, Christina M. Bebber, Yuqin Dai, Sarah Bopp, Anna Edenhofer, Albert Mao Li, Thomas N. Rösner, Lena Berning, Ming Yang, Logan B. Leak, Jenny Stroh, Bindesh Shrestha, Ali Taleb Abdallah, Dimitrios Prymidis, Hernando J. Olivos, , Julien Sage

The paper identifies metabolic differences between ASCL1-high and low SCLC cells, showing that ASCL1-high cells resist ferroptosis via BH4/BH2 antioxidants and are vulnerable to combined cysteine depletion and BH4/BH2 inhibition.

bioRxiv · Mar 31, 2026 · PreprintCancer Biology