Author

Sarah Bopp

Stanford Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Albert Mao LiiD· 1 shared Alexandros P. DrainasiD· 1 shared Ali Taleb AbdallahiD· 1 shared Àlvaro Quintanal-VillalongaiD· 1 shared Ana Cetkovic· 1 shared Andrea C. ChaikovskyiD· 1 shared Angela RiedeliD· 1 shared Anna Edenhofer· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Albert Mao LiORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

MYC/MIZ1 suppression of lysosomal protein degradation drives immune evasion in pancreatic ductal adenocarcinoma

Bastian Krenz, Toshitha Kannan, Greta Mattavelli, Werner Schmitz, Anneli Gebhardt-Wolf, Mara John, Ana Cetkovic, Arpa Aintablian, Stella Caviccioli, Lukas Nowak, Sarah Bopp, Christina Schuelein‐Voelk, Carsten Patrick Ade, Steffi Herold, Julian Bender, , Martin Eilers

This study shows that MYC suppresses lysosomal protein degradation in pancreatic ductal adenocarcinoma, promoting immune evasion by altering amino acid availability and immune cell responses.

bioRxiv · Jul 23, 2026 · PreprintCancer Biology

Cell state-specific metabolic networks govern ferroptosis versus apoptosis in small cell lung cancer

Jun W. Kim, Christina M. Bebber, Yuqin Dai, Sarah Bopp, Anna Edenhofer, Albert Mao Li, Thomas N. Rösner, Lena Berning, Ming Yang, Logan B. Leak, Jenny Stroh, Bindesh Shrestha, Ali Taleb Abdallah, Dimitrios Prymidis, Hernando J. Olivos, , Julien Sage

The paper identifies metabolic differences between ASCL1-high and low SCLC cells, showing that ASCL1-high cells resist ferroptosis via BH4/BH2 antioxidants and are vulnerable to combined cysteine depletion and BH4/BH2 inhibition.

bioRxiv · Mar 31, 2026 · PreprintCancer Biology