Author

E. F. Griffin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

C. S. Kyriakis· 2 shared E. W. Howerth· 2 shared S. M. Tompkins· 2 shared A. Fasano· 1 shared A. M. Lozano· 1 shared C. Burns· 1 shared C. L. Siepker· 1 shared C. S. Asensio· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

C. S. Kyriakis

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pandemic Risk Assessment for a Swine Influenza A Virus in Comparative Human Substrates

I. Padykula, L. Damodaran, K. T. Young, M. Krunkosky, E. F. Griffin, J. F. North, P. J. Neasham, V. C. Pleiasis, C. L. Siepker, J. B. Stanton, E. W. Howerth, J. Bahl, C. S. Kyriakis, S. M. Tompkins

The paper assesses a swine influenza A virus in animal and human cell models to evaluate pandemic risk, finding varied replication potential across models.

bioRxiv · Feb 24, 2024 · PreprintMicrobiology

Swine influenza A virus isolates containing the pandemic H1N1 origin matrix gene elicit greater disease in the murine model

S. J. Curran, E. F. Griffin, L. Ferreri, C. S. Kyriakis, E. W. Howerth, D. R. Perez, S. M. Tompkins

This study shows that swine influenza A viruses with the pandemic H1N1 origin matrix gene cause more severe disease in mice compared to those with the classical swine matrix gene.

bioRxiv · Sep 25, 2023Microbiology

VPS41 recessive mutation causes ataxia and dystonia with retinal dystrophy and mental retardation by inhibiting HOPS function and mTORC1 signaling

R. E. N. van der Welle, R. Jobling, C. Burns, P. Sanza, C. ten Brink, A. Fasano, L. Chen, F. J. Zwartkruis, S. Zwakenberg, E. F. Griffin, J. van der Beek, T. Veenendaal, N. Liv, S. Blaser, C. Sepulveda, , J. Klumperman

This paper identifies VPS41 mutations causing neurodegenerative disease by disrupting HOPS function and mTORC1 signaling, leading to lysosomal dysfunction and impaired autophagy.

bioRxiv · Dec 18, 2019 · PreprintCell Biology