Author

Fatimah H Hussein

Wayne State University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:Drosophila· 2 Assay:proteomics· 2 Approach:statistical model· 2 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Sokol V. TodiiD· 3 shared Kristin RichardsoniD· 2 shared Matthew V. PriftiiD· 2 shared Truman Kula· 2 shared Anna StephaniD· 1 shared Anthony A. HighiD· 1 shared Bedri RanxhiiD· 1 shared Fabio DemontisiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Sokol V. TodiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Androgen Receptor SUMO site mutations ameliorate toxicity in Drosophila models of SBMA

Kristin Richardson, Matthew V. Prifti, Truman Kula, Fatimah H Hussein, Sokol V. Todi

This study shows that mutating SUMOylation sites in the androgen receptor in Drosophila models of SBMA improves longevity but has limited effects on motor performance.

bioRxiv · Oct 24, 2026 · PreprintNeuroscience

Polyglutamine-expanded TBP rewires neuronal and muscle proteomes beyond transcription in a Drosophila model of SCA17

Matthew V. Prifti, Kristin Richardson, Fatimah H Hussein, Truman Kula, Anna Stephan, Yong-Dong Wang, Wei Wang, Anthony A. High, Suresh Poudel, Fabio Demontis, Sokol V. Todi

This study identifies tissue-specific proteomic changes in a Drosophila SCA17 model, showing polyglutamine-expanded TBP affects pathways beyond transcription.

bioRxiv · Oct 21, 2026 · PreprintNeuroscience

Chronic alpha-Synuclein Over-Expression and Ceruloplasmin Challenge Promote Distinct Iron and Redox Responses in M17 Cells

Masoumeh Pourhadi, Bedri Ranxhi, Seth P Sukaria, Fatimah H Hussein, Sokol V. Todi, Peter A. LeWitt, Wei‐Ling Tsou

This study investigates how chronic alpha-synuclein over-expression and ceruloplasmin-mediated iron modulation affect iron and redox homeostasis in neuroblastoma cells.

bioRxiv · Jun 16, 2026 · PreprintNeuroscience