Author

H. MacMillan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 3 Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 5 Organism:bacteria (other)· 3 Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Assay:proteomics· 3 Research Focus:development & morphogenesis· 2 Research Focus:differential expression· 2 Organism:human· 2

Frequent Co-Authors

A. Persat· 3 shared R. Patino· 3 shared Y. Inclan· 3 shared A. Talla· 2 shared E. M. Coffey· 2 shared E. W. Newell· 2 shared J. L. Czartoski· 2 shared J. N. Engel· 2 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMicrobiology

A. Persat

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

Spatial control of sensory adaptation modulates mechanosensing in Pseudomonas aeruginosa

R. Patino, M. J. Kuhn, H. Macmillan, Y. Inclan, I. Chavez, J. Von Dollen, J. R. Johnson, D. L. Swaney, N. Krogan, A. Persat, J. Engel

This study identifies a spatial mechanism of sensory adaptation in Pseudomonas aeruginosa, revealing how enzyme localization modulates mechanosensing and virulence.

bioRxiv · Feb 27, 2024 · PreprintMicrobiology

Pseudomonas aeruginosa mechanosensing controls cell polarity during twitching by activating two antagonistic response regulators

M. J. Kuhn, H. Macmillan, L. Tala, Y. Inclan, R. Patino, X. Pierrat, Z. Al-Mayyah, J. N. Engel, A. Persat

This study identifies how two response regulators, PilG and PilH, control cell polarity in Pseudomonas aeruginosa during twitching motility by antagonistically responding to mechanosensing signals.

bioRxiv · Nov 15, 2022Microbiology

Persistent serum protein signatures define an inflammatory subset of long COVID

A. Talla, S. V. Vasaikar, G. Szeto, M. P. Lemos, J. L. Czartoski, H. MacMillan, Z. Moodie, K. W. Cohen, L. B. Fleming, Z. Thomson, L. Okada, L. A. Becker, E. M. Coffey, S. C. DeRosa, E. W. Newell, , T. R. Torgerson

Serum proteome profiling identifies subsets of long COVID patients with evidence of persistent inflammation and key immune signaling pathways.

bioRxiv · May 9, 2022 · PreprintImmunology

Longitudinal immune dynamics of mild COVID-19 define signatures of recovery and persistence

A. Talla, S. V. Vasaikar, M. P. Lemos, Z. Moodie, M.-P. L. Pebworth, K. E. Henderson, K. W. Cohen, J. L. Czartoski, L. Lai, M. S. Suthar, A. T. Heubeck, P. C. Genge, C. R. Roll, M. Weiss, J. Reading, , T. F. Bumol

The paper profiles longitudinal immune responses in mild COVID-19 patients to identify recovery and persistence signatures.

bioRxiv · May 26, 2021 · PreprintImmunology

Mechanotaxis directs Pseudomonas aeruginosa twitching motility.

L. Tala, M. Kuhn, Y. Inclan, R. Patino, X. Pierrat, I. Vos, Z. Al-Mayyah, H. MacMillan, J. Negrete, J. N. Engel, A. Persat

This study identifies mechanotaxis as the mechanism by which Pseudomonas aeruginosa directs twitching motility using the Chp system to control directional migration.

bioRxiv · Jan 26, 2021Microbiology

Chilling induces unidirectional solute leak through the locust gut epithelia

K. Brzezinski, H. MacMillan

This study shows that chilling causes unidirectional solute leak through the locust gut epithelia, specifically from mucosal to serosal direction, but not the reverse.

bioRxiv · Sep 26, 2019Physiology