Author

I. Sermet-Gaudelus

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Edelman· 2 shared A. Hinzpeter· 2 shared A. Charbit· 1 shared A. Delaunay-Moisan· 1 shared A. Ferroni· 1 shared A. Golec· 1 shared A. Hadchouel Duverge· 1 shared A. Jamet· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

A. Edelman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Keratin 8 is a scaffolding and regulatory protein of ERAD complexes

I. M. Pranke, B. Chevalier, A. Premchandar, N. Baatallah, K. F. Tomaszewski, S. Bitam, D. Tondelier, A. Golec, J. Stolk, G. L. Lukacs, P. S. Hiemstra, M. Dadlez, D. A. Lomas, J. A. Irving, A. Delaunay-Moisan, , A. Edelman

This study shows that Keratin 8 modulates ERAD complexes, enhancing degradation of misfolded proteins like Z-A1AT and F508del-CFTR, and suggests targeting K8 for therapeutic intervention in A1ATD.

bioRxiv · Feb 1, 2022Cell Biology

Elexacaftor/Tezacaftor/Ivacaftor alters branching morphogenesis of the mouse embryonic lung

M. Lhuillier, L. Aoust, E. Dreano, M.-L. Franco-Montoya, K. Landry-Truchon, N. Houde, S. Chhun, A. Hinzpeter, A. Edelman, C. Delacourt, L. Jeannotte, I. Sermet-Gaudelus, A. Hadchouel Duverge

This paper shows that the triple CFTR modulator therapy ETI reduces lung branching and increases terminal bud dilation in mouse embryos, suggesting potential developmental risks.

bioRxiv · Nov 1, 2021 · PreprintPharmacology & Toxicology

Chronic Staphylococcus aureus lung infection correlates with proteogenomic and metabolic adaptations leading to an increased intracellular persistence

X. TAN, M. Coureuil, E. Ramond, D. Euphrasie, M. Dupuis, F. Tros, J. Meyer, I. Nemanzny, C. Chhuon, I. C. Guerrera, A. Ferroni, I. Sermet-Gaudelus, X. Nassif, A. Charbit, A. Jamet

This study shows that S. aureus isolates from CF patients have increased intracellular persistence and metabolic adaptations, suggesting an intracellular niche contributes to chronic lung infection.

bioRxiv · Oct 16, 2018Microbiology