Author

J. E. Peters

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Evolutionary Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:bacteria (other)· 2 Approach:comparative / phylogenetic analysis· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

A. D. Grossman· 1 shared A. Guarne· 1 shared B.-M. Koo· 1 shared C. A. Gross· 1 shared C. C. Hearne· 1 shared C. H. S. Lu· 1 shared E. V. Koonin· 1 shared G. E. Heussler· 1 shared

Activity by Year

2017-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperMicrobiology

A. D. Grossman

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Author profile →

3 papers

Recent·Most cited

Structural basis for DNA targeting by the Tn7 transposon

Y. Shen, J. Gomez-Blanco, M. T. Petassi, J. E. Peters, J. Ortega, A. Guarne

This study reveals the structural mechanism of Tn7 transposon target-site selection using cryo-EM, showing how TnsC distinguishes target DNA and prevents reinsertion.

bioRxiv · May 24, 2021Biophysics

Mobile-CRISPRi: Enabling Genetic Analysis of Diverse Bacteria

J. M. Peters, B.-M. Koo, R. Patino, G. E. Heussler, C. C. Hearne, Y. Inclan, J. S. Hawkins, C. H. S. Lu, M. M. Harden, H. Osadnik, J. E. Peters, J. N. Engel, R. J. Dutton, A. D. Grossman, C. A. Gross, O. S. Rosenberg

The paper introduces Mobile-CRISPRi, a CRISPRi system for non-model bacteria, demonstrating its use in analyzing gene function and antibiotic resistance in human pathogens.

bioRxiv · May 5, 2018Microbiology

Recruitment of CRISPR-Cas systems by Tn7-like transposons

J. E. Peters, K. S. Makarova, S. Shmakov, E. V. Koonin

The paper identifies CRISPR-Cas systems within Tn7-like transposons, showing they are truncated and function in pre-crRNA processing but not target cleavage, and proposes a mechanism for their role in transposon spread.

bioRxiv · Jun 23, 2017Evolutionary Biology