Author

J. Harder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. Chiramel· 1 shared A. Shamsaddini· 1 shared B. T. Emmer· 1 shared C. Baker· 1 shared C. Brunton· 1 shared C. Clancy· 1 shared C. E. Wobus· 1 shared C. Martens· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

A. Chiramel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ARF6 is a host factor for SARS-CoV-2 infection in vitro

C. Mirabelli, E. J. Sherman, J. Bragazzi Cunha, J. W. Wotring, J. El Saghir, J. Harder, J. Z. Sexton, B. T. Emmer, C. E. Wobus

This study identifies ARF6 as a host factor critical for SARS-CoV-2 entry and infection in vitro, showing its importance across different cell models.

bioRxiv · Jun 9, 2022 · PreprintMicrobiology

Staphylococcus aureus activates the Aryl Hydrocarbon Receptor in Human Keratinocytes

E.-L. Stange, F. Rademacher, K. A. Drerup, N. Heinemann, L. Möbus, R. Gläser, J. Harder

This study shows that Staphylococcus aureus activates the Aryl Hydrocarbon Receptor (AhR) in human keratinocytes, leading to cytokine production and highlighting AhR as a novel sensor for S. aureus.

bioRxiv · Jan 5, 2022 · PreprintImmunology

Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19

S. Robertson, O. Bedard, K. McNally, C. Shaia, C. Clancy, M. Lewis, R. Broeckel, A. Chiramel, J. G. Shannon, G. Sturdevant, R. Rosenke, S. L. Anzick, E. Forte, C. Preuss, C. Baker, , S. Best

The paper uses genetically diverse mice to model varied innate immune responses to SARS-CoV-2 infection, showing differences in survival, viral replication, and cytokine profiles.

bioRxiv · Sep 17, 2021Microbiology