Author

Jonathan Moss

MRC Centre for Regenerative Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

Anna WilliamsiD· 2 shared Ami Onishi· 1 shared Angus LawsoniD· 1 shared Anna J. StevensoniD· 1 shared Antoine VallatosiD· 1 shared Balazs Viktor VargaiD· 1 shared Barry W. McColliD· 1 shared Blanca Díaz CastroiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersNeuroscience

Anna WilliamsORCID

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Author profile →

3 papers

Recent·Most cited

Multiregional blood-brain barrier phenotyping identifies the prefrontal cortex as the most vulnerable region to ageing in mice

Isabel Bravo‐Ferrer, Katrine Gaasdal-Bech, Chiara Colvin, Hollie J Vaughan, Jonathan Moss, Anna Williams, Blanca Díaz Castro

The paper identifies the prefrontal cortex as the most vulnerable BBB region to ageing in mice by analyzing molecular, structural, and functional changes across three brain regions.

Brain Communications · Jan 1, 2025Peer-reviewedNeuroscience

Human astrocytes and microglia show augmented ingestion of synapses in Alzheimer's disease via MFG-E8

Makis Tzioras, Michael J. D. Daniels, Caitlin Davies, Paul S. Baxter, Declan King, Sean McKay, Balazs Viktor Varga, Karla Popovic, Madison Hernandez, Anna J. Stevenson, Jack Barrington, Elizabeth Drinkwater, Julia Borella, Rebecca K. Holloway, Jane Tulloch, , Tara L. Spires‐Jones

The paper shows human astrocytes and microglia ingest more synapses in Alzheimer's disease via MFG-E8, and inhibiting MFG-E8 reduces this ingestion in cultured cells.

Cell Reports Medicine · Aug 30, 2023Peer-reviewedNeuroscience

An intrinsic endothelial dysfunction causes cerebral small vessel disease

Sophie Quick, Tessa V. Procter, Jonathan Moss, Angus Lawson, Serena Baker, Marc Walton, Mehreen Mohammad, Will Mungall, Ami Onishi, Zuzanna Tobola, Michael S. Stringer, Maurits A. Jansen, Antoine Vallatos, Ylenia Giarratano, Miguel O. Bernabéu, , Anna Williams

This study identifies intrinsic endothelial dysfunction as a cause of cerebral small vessel disease in a normotensive rat model, independent of hypertension.

bioRxiv · Dec 13, 2021 · PreprintNeuroscience