Author

Julia Kleniuk

University of Cambridge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Evan ReidiD· 2 shared Aishwarya G. NadadhuriD· 1 shared Catherine E. Rodger· 1 shared Denisa HathaziiD· 1 shared Dorota Pregowska· 1 shared Eliska ZlamalovaiD· 1 shared Emily Wolfenden· 1 shared Grant W. A. Neilson· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersNeuroscience

Evan ReidORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell-type-specific adaptations to mitochondrial stress underly the neurological presentations of MTRFR mutations

Mariana Zarate-Mendez, Kaela O’Connor, Natalia Malig, Oliver Podmanicky, Julia Kleniuk, Sally Spendiff, Evan Reid, Hanns Lochmüller, Denisa Hathazi, Rita Horváth

This study identifies cell-type-specific adaptations to mitochondrial stress in MTRFR mutations, showing cortical neurons activate survival programs while motor neurons undergo apoptosis.

bioRxiv · Dec 31, 2025 · PreprintNeuroscience

Protrudin acts at ER-endosome contacts to promote KIF5-mediated endosomal fission and endosome-to-Golgi transport

Julia Kleniuk, Aishwarya G. Nadadhur, Catherine E. Rodger, Emily Wolfenden, Isabelle A. Hall, Samuel R. Cheers, Eliska Zlamalova, Evan Reid

This study identifies protrudin's role in promoting endosomal tubule fission and endosome-to-Golgi transport by interacting with ER-localised VAP proteins and KIF5.

Neurobiology of Disease · Dec 20, 2025Peer-reviewedCell Biology

TP53 and RB1 are predictive genetic biomarkers for sensitivity to cytarabine in gliomas

Liisi Laaniste, Joachim Luginbühl, Mi K. Trinh, Rachel Watkins, Gregor Lueg, Grant W. A. Neilson, Marjolein Burgers, Kasia Kania, Regina Hertfelder Reynolds, Zoe Smith, Julia Kleniuk, Lauren A. Gould, Dorota Pregowska, Sophie Mathiesen, Michael R. Johnson, Louwai Muhammed

The paper identifies TP53 and RB1 wild-type status as predictors of cytarabine sensitivity in gliomas using CRISPRi screening and machine learning analysis.

bioRxiv · Nov 20, 2025 · PreprintCancer Biology