Author

K. Onishi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Organism:mouse· 2 Research Focus:drug response / drug discovery· 1 Organism:fungi (other)· 1 Research Focus:host-pathogen & immune response· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

F. Li· 2 shared J. H.-C. Wang· 2 shared J. Zhang· 2 shared A. Watanabe· 1 shared D. Hagiwara· 1 shared D. Nie· 1 shared G. Zhao· 1 shared H. Takahashi· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

F. Li

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Functionally conserved Pkd2, mutated in autosomal dominant polycystic kidney disease, localizes to the endoplasmic reticulum and regulates cytoplasmic calcium homeostasis in fission yeast

T. Koyano, K. Kume, K. Onishi, M. Matsuyama, M. Fukushima, T. Toda

This study shows that Pkd2, a homolog of human polycystin-2, localizes to the ER and regulates calcium homeostasis in fission yeast, with functional conservation between species.

bioRxiv · Sep 20, 2022 · PreprintCell Biology

Platelet HMGB1 in Platelet-Rich Plasma Promotes Tendon Wound Healing

J. Zhang, F. Li, T. Augi, K. M. Williamson, K. Onishi, M. V. Hogan, M. D. Neal, J. H.-C. Wang

This study shows that platelet HMGB1 in PRP enhances tendon healing in mice, with PRP from GFP mice promoting faster healing and better tissue organization compared to PRP from HMGB1 KO mice.

bioRxiv · Apr 22, 2021Cell Biology

Identification of novel mutations contributing to azole tolerance of Aspergillus fumigatus through in vitro exposure to tebuconazole

T. Toyotome, K. Onishi, M. Sato, Y. Kusuya, D. Hagiwara, A. Watanabe, H. Takahashi

bioRxiv · Jul 20, 2020Microbiology

HMGB1 mediates the development of tendinopathy due to mechanical overloading

G. Zhao, J. Zhang, D. Nie, Y. Zhou, F. Li, K. Onishi, J. H.-C. Wang

This study shows that HMGB1 release in response to mechanical overloading causes inflammation and tendinopathy in mice, which can be blocked by glycyrrhizin.

bioRxiv · Aug 29, 2019Cell Biology