Author

T. Koyano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

M. Matsuyama· 4 shared M. Fukushima· 3 shared M. Namba· 2 shared T. Kobayashi· 2 shared A. Nishiyama· 1 shared D. Nakano· 1 shared J. Sun· 1 shared J. Wu· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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4 shared papersCell Biology

M. Matsuyama

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4 papers

Recent·Most cited

Functionally conserved Pkd2, mutated in autosomal dominant polycystic kidney disease, localizes to the endoplasmic reticulum and regulates cytoplasmic calcium homeostasis in fission yeast

T. Koyano, K. Kume, K. Onishi, M. Matsuyama, M. Fukushima, T. Toda

This study shows that Pkd2, a homolog of human polycystin-2, localizes to the ER and regulates calcium homeostasis in fission yeast, with functional conservation between species.

bioRxiv · Sep 20, 2022 · PreprintCell Biology

LRRC23 is a conserved component of the radial spoke that is necessary for sperm motility and male fertility in mice

X. Zhang, J. Sun, Y. Lu, J. Zhang, K. Shimada, T. Noda, S. Zhao, T. Koyano, M. Matsuyama, S. Zhou, J. Wu, M. Ikawa, M. Liu

This study identifies LRRC23 as a conserved component of the radial spoke complex in mouse sperm, essential for flagellar motility and male fertility but not ciliary function.

Journal of Cell Science · Oct 21, 2021Peer-reviewedCell Biology

Creation of X-linked Alport Syndrome Rat Model with Col4a5 Deficiency

M. Namba, T. Kobayashi, M. Kohno, T. Koyano, T. Hirose, M. Fukushima, M. Matsuyama

This study creates a novel Alport syndrome rat model with Col4a5 deficiency to better understand renal disease mechanisms and therapeutic targets.

bioRxiv · Apr 13, 2021Bioengineering

The p21 dependent G2 arrest of the cell cycle in epithelial tubular cells links to the early stage of renal fibrosis

T. Koyano, M. Namba, T. Kobayashi, K. Nakakuni, D. Nakano, M. Fukushima, A. Nishiyama, M. Matsuyama

The paper identifies p21-dependent G2 cell cycle arrest in proximal tubular cells as an early response to renal damage, which may protect kidney function during the initial stage of fibrosis.

bioRxiv · Feb 26, 2019Cell Biology