Author

Katharina Kladnik

Austrian Academy of Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1 Research Focus:transcription & RNA processing· 1

Frequent Co-Authors

Caroline SchätziD· 2 shared Fabian FrommeltiD· 2 shared Georg E. WinteriD· 2 shared Hana ImrichováiD· 2 shared Matthias HinterndorferiD· 2 shared Miquel Muñoz i OrdoñoiD· 2 shared Andrea RukavinaiD· 1 shared Anna C. ObenaufiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

Caroline SchätzORCID

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Author profile →

2 papers

Recent·Most cited

Induced Estrogen Receptor SUMOylation drives SERD activity

Matthias Hinterndorfer, Caroline Schätz, S. Schmitt, Martin Schoenlein, David M. Hoi, Izabela Kręcioch, Fabian Frommelt, Mykhailyna Shlei, Lukas Kater, Martin Pačesa, Miquel Muñoz i Ordoño, Georg Kempf, Katharina Kladnik, Paul Batty, Hana Imrichová, , Georg E. Winter

This study shows that SERDs induce ER SUMOylation, which recruits co-repressors to suppress transcription, rather than relying on ER degradation for their activity.

bioRxiv · Jun 26, 2026 · PreprintCancer Biology

Inhibitor-induced supercharging of kinase turnover via endogenous proteolytic circuits

Natalie S. Scholes, Martino Bertoni, Arnau Comajuncosa-Creus, Katharina Kladnik, Xuefei Guo, Fabian Frommelt, Matthias Hinterndorfer, Hlib Razumkov, Polina Prokofeva, Martin P. Schwalm, Florian Born, Sandra Röhm, Hana Imrichová, Brianda L. Santini, Eleonora Barone, , Georg E. Winter

The paper identifies inhibitor-induced kinase destabilization as a common phenomenon and reveals that it occurs through endogenous proteolytic mechanisms rather than classical ubiquitination.

Nature · Nov 26, 2025Peer-reviewedMolecular Biology