Author

Kayo Nishitani

National Center of Neurology and Psychiatry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Neuroscience· 4 Developmental Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Approach:statistical model· 4 Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:human· 3 Organism:mouse· 3 Assay:spatial transcriptomics· 3

Frequent Co-Authors

Mikio HoshinoiD· 5 shared Satoshi MiyashitaiD· 5 shared Kazumi ShimaokaiD· 4 shared Nao K N Tabe· 4 shared Kanako Komatsu· 3 shared Kaoru YagitaiD· 3 shared Keiya IijimaiD· 3 shared Kumiko Murayama· 3 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

5 shared papersNeuroscience

Mikio HoshinoORCID

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Spatial transcriptomics identifies L6 corticothalamic and L6b subplate-like heterotopic neurons in an epileptogenic SLC35A2-associated brain malformation.

Nao K N Tabe, Satoshi Miyashita, Kaoru Yagita, Keiya Iijima, Kazumi Shimaoka, Ai Hosaka, Sota Okumiya, Kayo Nishitani, Kanako Komatsu, Emi Usukura, Kumiko Murayama, Terunori Sano, Masaki Takao, Masaki Iwasaki, Mikio Hoshino

The paper identifies molecular features of WM heterotopic neurons in MOGHE using spatial transcriptomics and supports findings with immunohistochemistry and a mouse model.

bioRxiv · Sep 8, 2026 · PreprintNeuroscience

Comprehensive transcriptomic and proteomic profiling of mTOR-related epilepsy

Kaiyuan Ji, Satoshi Miyashita, Keiya Iijima, Kaoru Yagita, Tomoo Owa, Kazumi Shimaoka, Nao K N Tabe, Minami Mizuno, Ai Hosaka, Kayo Nishitani, Kumiko Murayama, Kanako Komatsu, Masaki Sone, Terunori Sano, Shinichiro Taya, , Mikio Hoshino

The paper integrates transcriptomic and proteomic data from mTOR-related epilepsy patients to identify impaired oxidative phosphorylation as a molecular hallmark.

bioRxiv · Mar 11, 2026 · PreprintNeuroscience

Single-cell-resolved spatial multi-omics identifies mTOR-driven neuronal and astroglial pathogenicity underlying epileptogenic focal cortical dysplasia

Kazumi Shimaoka, Satoshi Miyashita, Keiya Iijima, Kaoru Yagita, Nao K N Tabe, Koichi Hashizume, Nariko Arimura, Shuyu He, Minami Mizuno, Kayo Nishitani, Kanako Komatsu, Kumiko Murayama, Masaki Sone, Emi Usukura, Terunori Sano, , Mikio Hoshino

The paper uses spatial multi-omics to analyze FCDIIb tissue, revealing cell-type-specific effects of mTOR mutations on metabolic and immune pathways in drug-resistant epilepsy.

bioRxiv · Jan 23, 2026 · PreprintNeuroscience

Lineage Analysis and Molecular Characterization of Bergmann Glia-like Progenitors in the Postnatal Mouse Cerebellum using in vivo Electroporation and Spatial Transcriptomics

Kyoka Suyama, Toma Adachi, Minami Mizuno, Kaiyuan Ji, Eriko Isogai, Ikuko Hasegawa, Kayo Nishitani, Masaki Sone, Satoshi Miyashita, Tomoo Owa, Mikio Hoshino

This study characterizes Bergmann glia-like progenitors (BGLPs) in the postnatal mouse cerebellum using electroporation and spatial transcriptomics, revealing their developmental dynamics and gene expression profiles.

bioRxiv · May 20, 2025 · PreprintDevelopmental Biology

AUTS2, a causative gene for microcephaly, regulates division of intermediate progenitor cells and production of upper-layer neurons

Kazumi Shimaoka, Kei Hori, Satoshi Miyashita, Yukiko Inoue, Nao K N Tabe, Asami Sakamoto, Ikuko Hasegawa, Kayo Nishitani, Kunihiko Yamashiro, Saki F. Egusa, Shoji Tatsumoto, Yasuhiro Go, Manabu Abe, Kenji Sakimura, Takayoshi Inoue, , Mikio Hoshino

This study identifies AUTS2's role in regulating intermediate progenitor cell division and upper-layer neuron production through interaction with PRC2 and repression of genes like Robo1.

The EMBO Journal · Jan 15, 2025Peer-reviewedNeuroscience