Author

Kyle Tengler

The University of Texas at San Antonio Health Science Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

Justin W. C. LeungiD· 2 shared Douglas R. WalkeriD· 1 shared Elisar J. BarbariD· 1 shared Gargi GhosaliD· 1 shared Jesse Howe· 1 shared Kevin D. RaneyiD· 1 shared Kirk L. WestiD· 1 shared Maya Sonpatki· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiophysics

Justin W. C. LeungORCID

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2 papers

Recent·Most cited

LC8 enhances 53BP1 foci through heterogeneous bridging of 53BP1 oligomers

Jesse Howe, Douglas R. Walker, Kyle Tengler, Maya Sonpatki, Patrick N. Reardon, Justin W. C. Leung, Elisar J. Barbar

This study shows that LC8 bridges 53BP1 trimers to enhance 53BP1 foci formation, which is critical for DNA repair.

eLife · Apr 29, 2025Peer-reviewedBiophysics

Autophosphorylation of the Tousled-like kinases TLK1 and TLK2 regulates recruitment to damaged chromatin via PCNA interaction

Kirk L. West, Tram T N Nguyen, Kyle Tengler, Natasha F. Kreiling, Kevin D. Raney, Gargi Ghosal, Justin W. C. Leung

This study shows that TLK1 and TLK2 hyper-autophosphorylation suppresses their recruitment to damaged chromatin by masking the PIP-box, which is required for PCNA interaction.

Nucleic Acids Research · Dec 13, 2024Peer-reviewedGenetics