Author

M. C. Weiser-Evans

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

R. A. Nemenoff· 3 shared A. M. Johnson· 2 shared E. T. Clambey· 2 shared S. Lu· 2 shared A. J. Jolly· 1 shared A. J. Neuwelt· 1 shared A. K. Kimball· 1 shared A. M. Dubner· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

R. A. Nemenoff

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Author profile →

4 papers

Recent·Most cited

Smooth muscle-derived adventitial progenitor cells promote key cell type transitions controlling plaque stability in atherosclerosis in a Klf4-dependent manner

M. C. Weiser-Evans, A. M. Dubner, S. Lu, A. J. Jolly, K. A. Strand, M. F. Mutryn, T. Hinthorn, T. Noble, R. A. Nemenoff, K. S. Moulton, M. W. Majesky

This study shows that Klf4-dependent transitions in adventitial progenitor cells affect plaque stability in atherosclerosis by altering fibroblast, SMC, and macrophage phenotypes.

bioRxiv · Jul 18, 2023 · PreprintPathology

The TNF{blacktriangleup}ARE mouse as a model of intestinal fibrosis

C. A. Steiner, S. D. Koch, T. Evanoff, N. Welch, R. Kostelecky, R. Callahan, E. M. Murphy, C. H. Hall, S. Lu, M. C. Weiser-Evans, I. M. Cartwright, S. P. Colgan

The paper evaluates the TNF{Delta}ARE mouse as a model of intestinal fibrosis, finding it develops structural, histological, and genetic changes of ileal fibrosis by 24 weeks.

bioRxiv · Jan 13, 2023 · PreprintCell Biology

Adaptive immunity is required for durable responses to alectinib in murine models of EML4-ALK lung cancer

E. K. Kleczko, T. K. Hinz, T. T. Nguyen, N. J. Gurule, A. Navarro, A. T. Le, A. M. Johnson, J. Kwak, D. I. Polhac, E. T. Clambey, M. C. Weiser-Evans, T. Patil, E. L. Schenk, L. E. Heasley, R. A. Nemenoff

This paper shows that adaptive immunity enhances the durability of alectinib response in EML4-ALK lung cancer models, with CD8+ T cells playing a key role.

bioRxiv · Apr 14, 2022 · PreprintCancer Biology

Tumor-Intrinsic Response to IFNgamma Shapes the Tumor Microenvironment and Anti-PD-1 Response in NSCLC

B. L. Bullock, A. K. Kimball, J. M. Poczobutt, H. Y. Li, J. W. Kwak, A. J. Neuwelt, A. M. Johnson, E. Kleczko, R. Kaspar, K. Hopp, E. Schenk, M. C. Weiser-Evans, E. T. Clambey, R. A. Nemenoff

This study identifies tumor-intrinsic IFN-gamma signaling as a determinant of anti-PD-1 response in NSCLC by altering the tumor microenvironment.

bioRxiv · Jan 27, 2019Cancer Biology