Author

M. L. Kutys

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:mouse· 1

Frequent Co-Authors

K. A. Jacobs· 2 shared T. Singh· 2 shared A. Dema· 1 shared A. H. Cooper· 1 shared A. J. Hickey· 1 shared B. Griffith· 1 shared C. Cho· 1 shared C. P. Whitworth· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

K. A. Jacobs

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Doublecortin reinforces microtubules to promote growth cone advance in soft environments

A. Dema, R. A. Charafeddine, J. van Haren, S. Rahgozar, G. Viola, K. A. Jacobs, M. L. Kutys, T. Wittmann

This study shows DCX stabilizes microtubules in growth cones, enabling neuron morphogenesis in soft environments by resisting actomyosin contractile forces.

bioRxiv · Feb 28, 2024Cell Biology

Notch1 cortical signaling regulates epithelial architecture and cell-cell adhesion

M. J. White, K. A. Jacobs, T. Singh, M. L. Kutys

This study shows Notch1 cortical signaling, independent of transcription, regulates epithelial architecture and cell-cell adhesion by stabilizing adherens junctions and cortical actin.

bioRxiv · Jan 23, 2023 · PreprintCell Biology

Arp2/3 Complex Activity Enables Nuclear YAP for Naive Pluripotency of Human Embryonic Stem Cells

N. P. Meyer, T. Singh, M. L. Kutys, T. Nystul, D. L. Barber

This study identifies Arp2/3 complex activity as essential for nuclear YAP translocation and naive pluripotency in human embryonic stem cells through actin remodeling and Hippo signaling regulation.

bioRxiv · Jan 16, 2023Cell Biology

Microphysiological vascular malformation model reveals a role of dysregulated Rac1 and mTORC1/2 in lesion formation.

W. Y. Aw, C. Cho, H. Wang, A. H. Cooper, E. L. Doherty, D. Rocco, S. A. Huang, S. Kubik, C. P. Whitworth, R. Armstrong, A. J. Hickey, B. Griffith, M. L. Kutys, J. Blatt, W. J. Polacheck

The paper develops a microfluidic model of PIK3CA-driven vascular malformations and identifies dysregulated Rac1/PAK and mTORC1/2 signaling as key factors in lesion formation.

bioRxiv · Sep 3, 2022 · PreprintBioengineering