Author

M. M. Lee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1

Frequent Co-Authors

A. B. Apolo· 1 shared A. R. Nickolls· 1 shared A. Roy· 1 shared B. S. Mallon· 1 shared B. Sallis· 1 shared C. E. Wright· 1 shared C. G. Bonnemann· 1 shared D. M. De Silva· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. B. Apolo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Autocrine Signaling by Receptor Tyrosine Kinases in Urothelial Carcinoma of the Bladder

Y. H. Lee, M. M. Lee, D. M. De Silva, A. Roy, C. E. Wright, T. K. Wong, R. Costello, O. Olaku, R. L. Grubb, P. K. Agarwal, A. B. Apolo, D. P. Bottaro

The paper identifies oncogenic alterations in receptor tyrosine kinases (TKs) and their association with poor survival in bladder cancer patients.

bioRxiv · Oct 15, 2020Cancer Biology

Protein arginine methyltransferase 5 promotes metastasis via enhancing EGFR transcription and modulating AKT1 activation by methylation

L. Huang, X.-O. Zhang, O. Verdejo-Torres, K. Wigglesworth, X. Sun, B. Sallis, D. Moon, T. Huang, E. Rozen, L. Zhang, G. Wang, J. M. Shohet, M. M. Lee, J. Q. Wu

This study shows PRMT5 promotes cancer metastasis by enhancing EGFR transcription and modulating AKT1 activation through methylation, leading to EMT and tumor cell invasion.

bioRxiv · Aug 12, 2020 · PreprintCancer Biology

Human embryoid bodies model basal lamina assembly and muscular dystrophy

A. R. Nickolls, M. M. Lee, K. Zukosky, B. S. Mallon, C. G. Bonnemann

This study uses human induced pluripotent stem cells to generate embryoid bodies that model basal lamina formation and dystroglycanopathy-related ECM defects in muscular dystrophies.

bioRxiv · Sep 10, 2019 · PreprintCell Biology