Author

M. Martin-Alonso

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:development & morphogenesis· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

M. J. Oudhoff· 3 shared H. T. Lindholm· 2 shared N. Parmar· 2 shared P. M. Vornewald· 2 shared A. Diez-Sanchez· 1 shared A. F. M. Altelaar· 1 shared A. G. Arroyo· 1 shared H. Lindholm· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

M. J. Oudhoff

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Postnatal intestinal epithelial maturation by LSD1 controls the small intestinal immune cell composition independently from the microbiota

A. Diez-Sanchez, H. T. Lindholm, P. M. Vornewald, J. Ostrop, N. Parmar, T. N. Shaw, M. Martin-Alonso, M. J. Oudhoff

This study shows that LSD1 in intestinal epithelial cells controls immune cell composition postnatally, independent of microbiota.

bioRxiv · Sep 8, 2023Immunology

Mmp17-deficient mice exhibit heightened goblet cell effector expression in the colon and increased resistance to chronic Trichuris muris infection

P. M. Vornewald, R. Forman, R. Yao, N. Parmar, H. T. Lindholm, M. Martin-Alonso, K. J. Else, M. J. Oudhoff

This study shows that Mmp17-deficient mice have increased goblet cell effector genes and resistance to Trichuris muris infection without changes in traditional immune modulators.

bioRxiv · Jul 10, 2023Immunology

Smooth muscle-specific MMP17 (MT4-MMP) defines the intestinal ECM niche

M. Martin-Alonso, H. Lindholm, S. Iqbal, P. Vornewald, S. Hoel, M. Damen, A. F. M. Altelaar, P. Katajisto, A. G. Arroyo, M. J. Oudhoff

The paper identifies MMP17 as a smooth muscle-specific factor critical for intestinal epithelial repair and stem cell niche regulation through its cleavage of PERIOSTIN and activation of YAP.

bioRxiv · Jun 18, 2020Cell Biology