Author

M. N. Cash

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

M. Salemi· 2 shared A. R. Patel· 1 shared A. Riva· 1 shared A. S. Ramirez-Mata· 1 shared A. Y. Vittor· 1 shared B. Rife Magalis· 1 shared C. Mavian· 1 shared C. N. Mavian· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

M. Salemi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Infectivity of three Mayaro Virus (Genus Alphavirus, Family Togaviridae) geographic isolates in human cell lines

A. R. Patel, M. Dulcey, N. Abid, M. N. Cash, J. Dailey, M. Salemi, C. Mavian, A. Y. Vittor

This study compares the infectivity of three geographically distinct Mayaro virus isolates in human cell lines, finding one isolate exhibits faster infection kinetics and greater cytopathic effect due to mutations in the E2 protein.

bioRxiv · Sep 29, 2021 · PreprintMicrobiology

Brain tissue transcriptomic analysis of SIV-infected macaques identifies several altered metabolic pathways linked to neuropathogenesis, and Poly (ADP-ribose) polymerases (PARPs) as potential therapeutic targets

C. N. Mavian, A. S. Ramirez-Mata, J. J. Dollar, D. J. Nolan, K. White, S. N. Rich, B. Rife Magalis, M. N. Cash, S. Marini, M. Prosperi, D. Moraga Amador, A. Riva, K. C. Williams, M. Salemi

This study analyzes transcriptomic data from SIV-infected macaques to identify altered metabolic pathways and PARPs as potential therapeutic targets in HIV-associated neurodegeneration.

bioRxiv · May 21, 2020Pathology

Harmine enhances the activity of the HIV-1 latency-reversing agents ingenol A and SAHA

J. P. Taylor, L. H. Armitage, D. L. Aldridge, M. N. Cash, M. A. Wallet

This study shows that harmine enhances the efficacy of HIV-1 latency-reversing agents by increasing transcription factor availability and reducing HEXIM1 expression.

bioRxiv · Jan 31, 2020Microbiology