Author

M. O'Connor

University of Missouri–Kansas City

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Microbiology· 1 Molecular Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:proteomics· 3 Approach:statistical model· 3 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Assay:metagenomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Anandkumar· 1 shared A. Bogetti· 1 shared A. Brace· 1 shared A. Christiansen· 1 shared A. Clyde· 1 shared A. Dommer· 1 shared A. Flohr· 1 shared A. Gray· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiophysics

A. Anandkumar

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Resistance to antibacterial peptide nucleic acids through altered ribosome function

Jakob Frimodt-Møller, Irene Martinez-Roman, Thomas Prossliner, Lotta Happonen, Karen Leth Nielsen, Susanne Häußler, Elio Rossi, M. O'Connor, Peter Eigil Nielsen, Anders Løbner‐Olesen

This study identifies a ribosome-centered mechanism of resistance to antibacterial PNAs in E. coli, showing that mutations in ribosomal protein uS12 alter 30S assembly and translation-initiation dynamics, reducing PNA efficacy.

bioRxiv · Sep 9, 2026 · PreprintMolecular Biology

Elevated peripheral and nervous system inflammation is associated with decreased short-chain fatty acid levels in Zika-virus infected macaques

C. Miller, J. Manuzak, A. Gustin, C. Basting, R. Cheu, T. Schroeder, A. Velez, C. Driscoll, J. Tisoncik-Go, L. Schifanella, T. Hensley-McBain, C. Evandy, E. Smith, D. Bratt, J. Smedley, , N. R. Klatt

This study links reduced short-chain fatty acid levels with elevated inflammation in Zika-virus infected macaques, suggesting microbial dysfunction contributes to CNS and systemic inflammation.

bioRxiv · Jul 25, 2023 · PreprintMicrobiology

#COVIDisAirborne: AI-Enabled Multiscale Computational Microscopy of Delta SARS-CoV-2 in a Respiratory Aerosol

A. Dommer, L. Casalino, F. Kearns, M. Rosenfeld, N. Wauer, S.-H. Ahn, J. Russo, S. Oliveira, C. Morris, A. Bogetti, A. Trifan, A. Brace, T. Sztain, A. Clyde, H. Ma, , Muholland

This paper uses AI-enabled multiscale computational microscopy to study SARS-CoV-2 Delta variant in respiratory aerosols, revealing atomic-level insights into airborne transmission.

bioRxiv · Nov 12, 2021Biophysics

A Humanized Antibody against LRG1 that Inhibits Angiogenesis and Reduces Retinal Vascular Leakage

D. Kallenberg, V. Tripathi, F. Javaid, C. Pilotti, J. George, S. Davis, J. W. Blackburn, M. O'Connor, L. Dowsett, C. Bowers, S. E. Liyanage, M. Gourlaouen, A. Hoeh, F. Mota, D. Selwood, , S. E. Moss

bioRxiv · Jul 25, 2020 · PreprintPathology

EGFR Oncogenes Expressed In Glioblastoma Are Activated As Covalent Dimers And Paradoxically Stimulated By Erlotinib

E. Buck, M. O'Connor, D. Romashko, A. Salomatov, N. Ishiyama, D. Epstein, A. Mayweg, A. Flohr, R. Iacone, T. Nicolaides, S. Markovic, J. Zhang

The paper identifies a mechanism by which extracellular domain EGFR mutations in glioblastoma are activated through covalent dimerization, rendering ATP-site inhibitors ineffective and paradoxically promoting proliferation.

bioRxiv · Oct 21, 2019 · PreprintCancer Biology