Author

M. S. Dahabieh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Gramatica· 1 shared A. Talebi· 1 shared B. Samborska· 1 shared C. A. Scullion· 1 shared C. Goncalves· 1 shared C. M. Krawczyk· 1 shared D. G. Roy· 1 shared E. Battivelli· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperMicrobiology

A. Gramatica

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 9, 2023

13C metabolite tracing reveals glutamine and acetate as critical in vivo fuels for CD8+ T cells

E. H. Ma, M. S. Dahabieh, L. M. DeCamp…·R. G. Jones

This study uses 13C-labeled metabolites to show CD8+ T cells switch from glutamine to acetate metabolism during infection.

DOI 10.1101/2023.06.09.544407

mousemetabolomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintOct 25, 2022

Disrupting peroxisomes alters lipid metabolism in melanoma and uncovers a novel therapeutic vulnerability in combination with MAPK-targeted therapies

F. Huang, F. Cai, M. S. Dahabieh…·S. V. del Rincon

This paper identifies a metabolic vulnerability in melanoma cells by disrupting peroxisomes and shows that combining this with MAPK-targeted therapies enhances anti-tumor effects.

DOI 10.1101/2022.10.25.513718

humanbulk RNA-seq

𝕏 PostBluesky

MicrobiologybioRxivJan 4, 2018

Chromatin Functional States Correlate with HIV Latency Reversal in Infected Primary CD4+ T Cells

E. Battivelli, M. S. Dahabieh, M. Abdel-Mohsen…·E. Verdin

The paper identifies that latency reversing agents reactivate less than 5% of latent HIV proviruses and shows distinct chromatin functional states between reactivated and non-reactivated cells.

DOI 10.7554/elife.34655

humanbulk RNA-seq

𝕏 PostBluesky