Author

M. S. Stack

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

D. Martin· 1 shared E. Howe· 1 shared G. Bahcecioglu· 1 shared G. Deshpande· 1 shared H. Nakshatri· 1 shared I. Guldner· 1 shared K. L. Robert· 1 shared P. Zorlutuna· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBiochemistry

D. Martin

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Degradomic identification of membrane type 1-matrix metalloproteinase (MT1-MMP/MMP14) as an ADAMTS9 and ADAMTS20 substrate

S. Nandadasa, D. Martin, G. Deshpande, K. L. Robert, M. S. Stack, Y. Itoh, S. S. APTE

This study identifies ADAMTS9 and ADAMTS20 as substrates of MT1-MMP using N-terminomics, showing their role in regulating MT1-MMP activity and cell-surface function.

bioRxiv · Oct 17, 2022Biochemistry

Aged Breast Extracellular Matrix Drives Mammary Epithelial Cells to an Invasive and Cancer-Like Phenotype

G. Bahcecioglu, X. Yue, E. Howe, I. Guldner, M. S. Stack, H. Nakshatri, S. Zhang, P. Zorlutuna

This study shows that aged breast ECM drives mammary epithelial cells to an invasive, cancer-like phenotype through mechanisms involving EMT and LOX.

bioRxiv · Sep 30, 2020Cancer Biology