Author

E. Howe

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

S. Zhang· 2 shared A. K. Martino· 1 shared B. Palakurthi· 1 shared G. Bahcecioglu· 1 shared H. Nakshatri· 1 shared I. Guldner· 1 shared I. H. Guldner· 1 shared M. S. Stack· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

S. Zhang

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Maximizing the Anti-tumor Potential of Immune Checkpoint Blockade through Modulation of Myeloid-specific CXCL16 and STAT1 Signaling

B. Palakurthi, I. H. Guldner, X. Liu, A. K. Martino, Q. Wang, S. Fross, R. A. Neff, S. M. Golomb, E. Howe, S. Zhang

This study shows that modulating STAT1 signaling in chemotherapy-primed breast cancer enhances anti-PD-1 immunotherapy efficacy by altering myeloid cell dynamics.

bioRxiv · Mar 28, 2022 · PreprintCancer Biology

Aged Breast Extracellular Matrix Drives Mammary Epithelial Cells to an Invasive and Cancer-Like Phenotype

G. Bahcecioglu, X. Yue, E. Howe, I. Guldner, M. S. Stack, H. Nakshatri, S. Zhang, P. Zorlutuna

This study shows that aged breast ECM drives mammary epithelial cells to an invasive, cancer-like phenotype through mechanisms involving EMT and LOX.

bioRxiv · Sep 30, 2020Cancer Biology