Author

N. Kedia-Mehta

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

A. E. Hogan· 2 shared D. O'Shea· 2 shared L. V. Sinclair· 2 shared R. Bergin· 2 shared A. Berisha· 1 shared A. Woodcock· 1 shared B. M. Bradley· 1 shared C. DeBarra· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

A. E. Hogan

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2 papers

Recent·Most cited

Glycogen-fuelled metabolism supports rapid Mucosal Associated Invariant T cell responses

F. C. Cassidy, N. Kedia-Mehta, R. Bergin, A. Woodcock, A. Berisha, B. M. Bradley, E. Booth, O. K. Ryan, L. V. Sinclair, D. O'Shea, A. E. Hogan

This study shows glycogen metabolism supports rapid MAIT cell cytotoxicity and cytokine production, which may enhance their use in immunotherapy.

bioRxiv · Dec 20, 2022Immunology

Human Mucosal Associated Invariant T cell proliferation is dependent on a MYC-SLC7A5-Glycolysis metabolic axis

N. Kedia-Mehta, M. M. Pisarska, C. Rollings, C. O'Neill, C. DeBarra, C. Foley, N. A. Wood, N. Wrigley-Kelly, N. Veerapen, G. E. Besra, R. Bergin, N. Jones, D. O'Shea, L. V. Sinclair, A. E. Hogan

This study identifies a MYC-SLC7A5-Glycolysis metabolic axis critical for MAIT cell proliferation, showing its impairment in obesity.

bioRxiv · Jan 17, 2022 · PreprintImmunology