Author

R. B. Hamanaka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Y. Meliton· 3 shared K. A. Sun· 3 shared P. S. Woods· 3 shared G. M. Mutlu· 2 shared L. M. Kimmig· 2 shared O. R. Shamaa· 2 shared R. Cetin-Atalay· 2 shared Y. Tian· 2 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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3 shared papersCell Biology

A. Y. Meliton

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Mitochondrial One-Carbon Metabolism is Required for TGF-β-Induced Glycine Synthesis and Collagen Protein Production

A. Y. Meliton, R. Cetin-Atalay, Y. Tian, J. C. Szafran, K. W. D. Shin, T. Cho, K. A. Sun, P. S. Woods, O. R. Shamaa, B. Chen, A. Muir, G. M. Mutlu, R. B. Hamanaka

This study shows that mitochondrial one-carbon metabolism is essential for TGF-β-induced glycine synthesis and collagen production in lung fibroblasts, suggesting MTHFD2 inhibitors could treat IPF.

bioRxiv · Nov 7, 2023Cell Biology

HIF-1α induces glycolytic reprogramming in tissue-resident alveolar macrophagesto promote survival during acute lung injury

P. S. Woods, L. M. Kimmig, K. A. Sun, A. Y. Meliton, O. R. Shamaa, Y. Tian, R. Cetin-Atalay, W. W. Sharp, R. B. Hamanaka, G. M. Mutlu

This study shows hypoxia stabilizes HIF-1α in alveolar macrophages, promoting glycolytic reprogramming that enhances survival during acute lung injury.

bioRxiv · Feb 28, 2022Cell Biology

Endogenous itaconate is not required for particulate matter-induced NRF2 expression or inflammatory response

K. A. Sun, Y. Li, A. Y. Meliton, P. S. Woods, L. M. Kimmig, R. B. Hamanaka, G. Mutlu

This study shows endogenous itaconate is not required for PM-induced NRF2 expression or inflammation, but exogenous itaconate can suppress inflammation and activate NRF2.

bioRxiv · Jan 19, 2020Cell Biology