Author

R. K. Thomas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

M. L. Sos· 2 shared M. Peifer· 2 shared A. A. Hennrich· 1 shared A. Heimsoeth· 1 shared A. Placzek· 1 shared A. Schmitt· 1 shared C. Guo· 1 shared C. Lorenz· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

M. L. Sos

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Author profile →

3 papers

Recent·Most cited

Functional synapses between small cell lung cancer and glutamatergic neurons

A. Schmitt, V. Sakthivelu, K. Ndoci, G. A. Wani, M. Touet, I. Pintelon, I. Kisis, O. Ibruli, J. Weber, R. Maresch, C. M. Bebber, J. Goergens, M. Jevtic, F. Odenthal, A. Placzek, , F. Beleggia

This study identifies glutamatergic synaptic interactions between SCLC cells and neurons, showing that these interactions support tumor growth and can be targeted therapeutically.

bioRxiv · Jan 19, 2023Cancer Biology

Identification of TAZ as the essential molecular switch in orchestrating SCLC phenotypic transition and metastasis

h. Ji, Y. Jin, Q. Zhao, W. Zhu, Y. Feng, T. Xiao, P. Zhang, L. Jiang, Y. Hou, C. Guo, H. Huang, Y. Chen, X. Tong, J. Cao, X. Zhu, , L. Hu

The paper identifies TAZ as a molecular switch driving phenotypic transition and metastasis in SCLC by analyzing mouse models and human data.

bioRxiv · Jul 28, 2021Cancer Biology

In vitro model for resistance in oncogene-dependent tumors at the limit of radiological detectability

N. Müller, J. Brägelmann, C. Lorenz, U. P. Michel, D. Plenker, S. Ortiz-Cuaran, J. Weiss, R. Büttner, M. Peifer, R. K. Thomas, M. L. Sos, J. Berg

The paper develops an in vitro model to study resistance mechanisms in oncogene-dependent tumors and shows that combining drugs targeting multiple resistance pathways can inhibit resistant and drug-tolerant cell growth.

bioRxiv · Sep 5, 2019 · PreprintCancer Biology