Author

R. P. Galao

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Assay:metagenomics· 1 Organism:non-human primate· 1

Frequent Co-Authors

H. Wilson· 4 shared C. M. Swanson· 3 shared H. Winstone· 3 shared M. J. Lista· 3 shared A. Dyer· 2 shared A. H. Patel· 2 shared G. De Lorenzo· 2 shared G. Nebbia· 2 shared

Activity by Year

2021-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersMicrobiology

H. Wilson

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

The P681H mutation in the Spike glycoprotein escapes IFITM restriction and is necessary for type I interferon resistance in the SARS-CoV-2 alpha variant

M. J. Lista, H. Winstone, H. Wilson, A. Dyer, S. Pickering, R. P. Galao, G. De Lorenzo, V. Cowton, W. Furnon, N. M. Suarez, R. Orton, M. Palmirini, A. H. Patel, L. B. Snell, G. Nebbia, , S. J. Neil

This paper shows the P681H mutation in the SARS-CoV-2 alpha variant's Spike protein enables resistance to type I interferon and IFITM proteins, enhancing viral entry and transmission.

bioRxiv · Aug 11, 2022Microbiology

Regulation of KHNYN antiviral activity by the extended di-KH domain and nucleo-cytoplasmic trafficking

M. J. Lista, R. P. Galao, M. Ficarelli, D. Kmiec, H. Wilson, H. Winstone, E. R. Morris, H. E. Mischo, J. Wanford, R. Youle, C. Odendall, I. A. Taylor, S. J. Neil, C. Swanson

This paper identifies the extended di-KH and CUBAN domains in KHNYN as critical for its antiviral activity and interaction with ZAP, showing that the CUBAN domain regulates trafficking and abundance.

bioRxiv · Dec 22, 2021 · PreprintMicrobiology

The P681H mutation in the Spike glycoprotein confers Type I interferon resistance in the SARS-CoV-2 alpha (B.1.1.7) variant

M. J. Lista, H. Winstone, H. Wilson, A. Dyer, S. Pickering, R. P. Galao, G. De Lorenzo, V. Cowton, W. Furnon, N. M. Suarez, R. Orton, M. Palmarini, A. H. Patel, L. B. Snell, G. Nebbia, , S. Neil

This study shows the P681H mutation in the SARS-CoV-2 alpha variant's Spike protein confers resistance to Type I interferon, enhancing viral replication by evading innate immune responses.

bioRxiv · Nov 9, 2021 · PreprintMicrobiology

TRIM25 and ZAP target the Ebola virus ribonucleoprotein complex to mediate interferon-induced restriction

R. P. Galao, H. Wilson, K. L. Schierhorn, F. Debeljak, B. S. Bodmer, D. Goldhill, T. Hoenen, S. J. Wilson, C. M. Swanson, S. Neil

This study identifies TRIM25 and ZAP as key factors in type I interferon-mediated inhibition of Ebola virus replication by targeting the viral ribonucleoprotein complex.

bioRxiv · May 23, 2021Microbiology