Author

S. Iwano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:imaging· 3 Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:imputation· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

A. Miyawaki· 2 shared A. Kamiyama· 1 shared A. Yoshiki· 1 shared C. Oki· 1 shared F. Sugiyama· 1 shared H. Ichise· 1 shared H. Ito· 1 shared K. Abe· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersBioengineering

A. Miyawaki

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Akaluc bioluminescence offers superior sensitivity to track in vivo dynamics of SARS-CoV-2 infection

T. Tamura, H. Ito, S. Torii, L. Wang, S. Tsujino, A. Kamiyama, Y. Oda, Y. Morioka, S. Suzuki, K. Shirakawa, K. Sato, K. Yoshimatsu, Y. Matsuura, S. Iwano, S. Tanaka, T. Fukuhara

This study introduces AkaBLI, a method using Akaluc bioluminescence to track SARS-CoV-2 infection in vivo with higher sensitivity than NanoLuc, showing its utility in monitoring viral dynamics and vaccine efficacy.

bioRxiv · Oct 12, 2023 · PreprintMicrobiology

Development of two mouse strains conditionally expressing bright luciferases with distinct emission spectra as new tools for in vivo imaging

T. Nakashiba, K. Ogoh, S. Iwano, T. Sugiyama, S. Iijima-Mizuno, K. Nakashima, S. Mizuno, F. Sugiyama, A. Yoshiki, A. Miyawaki, K. Abe

This paper develops two mouse strains with Cre-dependent luciferases for in vivo imaging, showing distinct emission spectra and improved brightness for specific tissues.

bioRxiv · Apr 28, 2023 · PreprintBioengineering

Metastatic Single Tumor Cells Evade NK Cell-mediated Killing by Thrombin-mediated Loss of the Activating Ligand CD155/PVR/Necl-5

H. Ichise, S. Tsukamoto, T. Hirashima, Y. Konishi, C. Oki, S. Tsukiji, S. Iwano, A. Miyawaki, K. Sumiyama, K. Terai, M. Matsuda

The paper shows metastatic tumor cells evade NK cell killing by losing CD155/PVR/Necl-5 via thrombin, allowing survival in lung capillaries.

bioRxiv · Jan 15, 2021Immunology