Author

S. M. Greising

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

A. E. Qualls· 2 shared A. S. Nichenko· 2 shared J. A. Call· 2 shared W. M. Southern· 2 shared A. E. Thalacker-Mercer· 1 shared A. G. Schifino· 1 shared A. Yin· 1 shared H. Yin· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPhysiology

A. E. Qualls

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2 papers

Recent·Most cited

Lifelong Ulk1-mediated autophagy deficiency in muscle induces mitochondrial dysfunction and contractile weakness

A. S. Nichenko, J. R. Sorensen, W. M. Southern, A. E. Qualls, A. G. Schifino, J. McFaline-Figueroa, J. E. Blum, A. E. Thalacker-Mercer, S. M. Greising, J. A. Call

This study shows that lifelong Ulk1-mediated autophagy deficiency in muscle leads to mitochondrial dysfunction and contractile weakness in aging.

bioRxiv · Jul 31, 2020Physiology

PGC-1α overexpression partially rescues impaired oxidative and contractile pathophysiology following volumetric muscle loss injury

W. M. Southern, A. S. Nichenko, K. F. Tehrani, M. J. McGranahan, L. Krishnan, A. E. Qualls, N. J. Jenkins, L. J. Mortensen, H. Yin, A. Yin, R. E. Guldberg, S. M. Greising, J. A. Call

This study shows that PGC-1α overexpression improves oxidative capacity and muscle strength in VML-injured muscle, suggesting it as a potential therapeutic target.

bioRxiv · Jan 31, 2019Physiology