Author

S. M. Groves

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

V. Quaranta· 3 shared A. Oleskie· 1 shared A. Richmond· 1 shared A. S. Ireland· 1 shared B. L. Witt· 1 shared C. P. Whitney· 1 shared C. Yan· 1 shared D. Ayers· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersCancer Biology

V. Quaranta

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CXCR2 expression during melanoma tumorigenesis controls transcriptional programs that facilitate tumor growth

J. Yang, K. Bergdorf, C. Yan, W. Luo, S.-C. Chen, D. Ayers, Q. Liu, X. Liu, M. R. Boothby, S. M. Groves, A. Oleskie, X. Zhang, D. Maeda, J. Zebala, V. Quaranta, A. Richmond

This study shows that CXCR2 expression in melanoma progenitor cells drives tumor growth by altering transcriptional programs and immune responses.

bioRxiv · Feb 22, 2023 · PreprintCancer Biology

Involvement of epithelial-mesenchymal transition genes in small cell lung cancer phenotypic plasticity

S. M. Groves, N. Panchy, D. R. Tyson, L. A. Harris, V. Quaranta, T. Hong

This study examines the relationship between epithelial-mesenchymal transition (EMT) and SCLC subtype plasticity using transcriptome data from mouse models and human samples.

bioRxiv · Sep 9, 2022Cancer Biology

ASCL1 represses a latent osteogenic program in small cell lung cancer in multiple cells of origin

R. R. Olsen, D. W. Kastner, A. S. Ireland, S. M. Groves, K. Pozo, C. P. Whitney, M. R. Guthrie, S. J. Wait, D. Soltero, B. L. Witt, V. Quaranta, J. E. Johnson, T. G. Oliver

This study shows ASCL1 represses a latent osteogenic program in SCLC, promoting tumor initiation and preventing differentiation into bone tumors.

bioRxiv · Nov 11, 2020Cancer Biology