Author

S. Urbanczyk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Approach:genetic perturbation / CRISPR· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

D. Mielenz· 3 shared F. Golombek· 2 shared K. Castiglione· 2 shared S. Brodesser· 2 shared S. R. Schulz· 2 shared A. Bozec· 1 shared A. M. Siddiqui· 1 shared A. Schwab· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersImmunology

D. Mielenz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Quantitative proteomics and lipidomics of TFG-deficient B cells provide insights into mechanisms of autophagic flux and plasma cell biology

T. D. Steinmetz, L. Reimann, S. R. Schulz, S. Urbanczyk, J. Thomas, A.-K. Himmelreich, F. Golombek, K. Castiglione, S. Brodesser, B. Warscheid, D. Mielenz

This study uses proteomics and lipidomics to analyze TFG-deficient B cells, identifying TFG's role in autophagy, plasma cell biology, and lipid homeostasis.

bioRxiv · Sep 1, 2022 · PreprintImmunology

Metabolic breakdown of non-small cell lung cancers by mitochondrial HSPD1 targeting

B. Parma, V. Ramesh, P. N. Gollavilli, A. M. Siddiqui, L. Pinna, A. Schwab, S. Marshall, S. Zhang, C. Pilarsky, F. Napoli, M. Volante, S. Urbanczyk, D. Mielenz, H. D. Schroeder, M. Stemmler, , P. Ceppi

This study identifies HSPD1 as a therapeutic target in NSCLC, showing its disruption leads to metabolic breakdown and suppressed tumor growth.

bioRxiv · Mar 9, 2021Cancer Biology

Mitochondrial function is essential for humoral immunity by controlling flux of the TCA cycle, phosphatidic acid and mTOR activity in B cells

S. Urbanczyk, O. Baris, J. Hofmann, F. Golombek, K. Castiglione, X. Meng, A. Bozec, D. Mougiakakos, S. R. Schulz, W. Schuh, U. Schloetzer-Schrehardt, T. Steinmetz, S. Brodesser, R. J. Wiesner, D. Mielenz

This study shows that mitochondrial function is critical for B cell differentiation into plasma cells by regulating TCA cycle flux, phosphatidic acid levels, and mTOR activity.

bioRxiv · Jan 14, 2021 · PreprintImmunology