Author

C. Pilarsky

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Organism:human· 4 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Guglielmi· 1 shared A. M. Schlitter· 1 shared A. M. Siddiqui· 1 shared A. Malinova· 1 shared A. Ruzzenente· 1 shared A. Scarpa· 1 shared A. Schwab· 1 shared B. Parma· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Guglielmi

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Author profile →

4 papers

Recent·Most cited

ecDNA amplification of MYC drives intratumor copy-number heterogeneity and adaptation to stress in PDAC

A. Malinova, D. Schreyer, E. Fiorini, D. Pasini, M. Bevere, S. D'Agosto, S. Andreani, F. Lupo, L. Veghini, S. Grimaldi, S. Pedron, C. Nourse, R. Salvia, G. Malleo, A. Ruzzenente, , V. Corbo

bioRxiv · Sep 27, 2023Cancer Biology

GATA4 and GATA6 loss-of-expression is associated with extinction of the classical programme and poor outcome in pancreatic ductal adenocarcinoma

M. P. de Andres, R. Jackson, C. Pilarsky, A. M. Schlitter, E. Costello, W. Greenhalf, P. Ganeh, T. Knoesel, D. Palmer, P. Ruemmele, W. Weichert, M. Buechler, T. Hackert, J. P. Neoptolemos, N. Malats, , F. X. Real

This study shows that low GATA4 and GATA6 expression in pancreatic ductal adenocarcinoma (PDAC) is linked to worse patient outcomes and loss of the classical tumor phenotype.

bioRxiv · Aug 9, 2021Cancer Biology

Metabolic breakdown of non-small cell lung cancers by mitochondrial HSPD1 targeting

B. Parma, V. Ramesh, P. N. Gollavilli, A. M. Siddiqui, L. Pinna, A. Schwab, S. Marshall, S. Zhang, C. Pilarsky, F. Napoli, M. Volante, S. Urbanczyk, D. Mielenz, H. D. Schroeder, M. Stemmler, , P. Ceppi

This study identifies HSPD1 as a therapeutic target in NSCLC, showing its disruption leads to metabolic breakdown and suppressed tumor growth.

bioRxiv · Mar 9, 2021Cancer Biology

The presence of copy number variants in specific topologically associating domains has prognostic value in many cancer types

L. Li, N. Barth, C. Pilarsky, L. Taher

This study identifies prognostic TADs in cancer by analyzing their mutation patterns and structural perturbations across multiple cancer types.

bioRxiv · Sep 23, 2019 · PreprintCancer Biology