Author

Saurabh Khasnavis

AbbVie (United States)

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:benchmarking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Organism:mouse· 1

Frequent Co-Authors

Jan StöhriD· 2 shared Akash ModyiD· 1 shared Alban OrdureauiD· 1 shared Alex Laishu· 1 shared Ameya S KulkarniiD· 1 shared Andrea EastesiD· 1 shared Chao Tai· 1 shared Chengzhong WangiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Jan StöhrORCID

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Author profile →

2 papers

Recent·Most cited

Progression GWAS followed by functional characterization implicates E3 ubiquitin ligase TRIM2 as a potential genetic modifier of Parkinson's Disease progression

Ameya S Kulkarni, Daniel J. Apicco, Saleh Tamim, Lauren Gibilisco, Layan Nahlawi, Rachel McFarland Lucia, Sujana Ghosh, Samantha Lent, Yanyu Liang, Saurabh Khasnavis, LAURA B. SMITH, Joshua D. Stender, Priyanka Vijay, Cindy Zadikoff, J. Wade Davis, , Hyun Ji Noh

The paper performs a GWAS on PD progression metrics and identifies TRIM2 as a genetic modifier, showing its role in regulating neurofilament light and alpha-synuclein aggregation.

Scientific Reports · Sep 2, 2026Peer-reviewedGenetic & Genomic Medicine

Pharmacological PINK1 activation ameliorates Pathology in Parkinson's Disease models

R Chin, Rishi Rakhit, Dara Ditsworth, Chengzhong Wang, Johan Bartholoméüs, Song Liu, Akash Mody, Alex Laishu, Andrea Eastes, Chao Tai, Roy Y. Kim, Jessica Li, Steven Hansberry, Saurabh Khasnavis, Victoria A. Rafalski, , Nicholas T. Hertz

This study identifies MTK458, a PINK1 activator, which reduces α-synuclein pathology and pUb levels in Parkinson's disease models and correlates plasma pUb with disease progression.

bioRxiv · Feb 14, 2023 · PreprintNeuroscience