Author

T. C. Saido

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

T. Saito· 2 shared A. Balakrishnan· 1 shared A. Javidan· 1 shared D. Howatt· 1 shared D. Thiagarajan· 1 shared H. A. Uchida· 1 shared H. Jun· 1 shared H. Komano· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersBiochemistry

T. Saito

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Suppression of amyloid-β secretion from neurons by cis-9, trans-11-octadecadienoic acid, an isomer of conjugated linoleic acid

S. Hata, K. Kikuchi, K. Kano, H. Saito, Y. Sobu, S. Kinoshita, T. Saito, T. C. Saido, Y. Sano, H. Taru, J. Aoki, H. Komano, T. Tomita, S. Natori, T. Suzuki

This study shows that c9,t11 CLA reduces amyloid-beta production in neurons by altering BACE1 and APP localization, potentially protecting against neurodegeneration.

bioRxiv · Sep 13, 2020Biochemistry

Inducible Depletion of Calpain-2 Mitigates Abdominal Aortic Aneurysm in Mice

L. Muniappan, M. Okuyama, A. Javidan, D. Thiagarajan, W. Jiang, J. J. Moorleghen, L. Yang, A. Balakrishnan, D. Howatt, H. A. Uchida, T. C. Saido, V. Subramanian

This study shows that calpain-2 depletion reduces abdominal aortic aneurysm (AAA) progression and rupture in mice by preserving cytoskeletal proteins and ECM integrity.

bioRxiv · Apr 15, 2020Pathology

Disrupted remapping of place cells and grid cells in knock-in model of Alzheimer’s disease

H. Jun, S. Soma, T. Saito, T. C. Saido, K. M. Igarashi

This study shows that in a knock-in Alzheimer’s disease model, remapping of place and grid cells is disrupted, leading to impaired spatial memory circuits.

bioRxiv · Nov 1, 2019Pathology