Author

T. Nonaka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

M. E. Deerhake· 2 shared M. Inoue· 2 shared M. L. Shinohara· 2 shared N. Aggarwal· 2 shared W. E. Barclay· 2 shared A. Consiglio· 1 shared C. Zurzolo· 1 shared D. Matsuike· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

M. E. Deerhake

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Neuronal Autophagy Failure Drives α-Synuclein Transfer to Microglia to Outsource Aggregate Clearance

R. Chakraborty, P. Samella, V. Testa, J. Montero-Munoz, T. Nonaka, M. Hasegawa, A. Consiglio, C. Zurzolo

The paper shows that neuronal autophagy failure leads to α-synuclein aggregate transfer to microglia, which then degrade them more efficiently.

bioRxiv · Apr 19, 2024 · PreprintCell Biology

Structure and Function of Gli123 Involved in Mycoplasma mobile Gliding

D. Matsuike, Y. O. Tahara, T. Nonaka, H. N. Wu, T. Hamaguchi, H. Kudo, Y. Hayashi, M. Arai, M. Miyata

This study investigates the structure and function of Gli123, a surface protein essential for Mycoplasma mobile gliding, revealing its conformational changes and evolutionary implications.

bioRxiv · Sep 7, 2022Microbiology

AIM2 inflammasome activation in astrocytes occurs during the late phase of EAE

W. E. Barclay, M. E. Deerhake, M. Inoue, T. Nonaka, K. Nozaki, N. A. Luzum, N. Aggarwal, E. A. Miao, M. L. Shinohara

This study identifies AIM2 inflammasome activation in astrocytes during the late phase of EAE, distinct from traditional inflammasome-mediated inflammation.

bioRxiv · Oct 3, 2021 · PreprintImmunology

Dectin-1 limits central nervous system autoimmunity through a non-canonical pathway

M. E. Deerhake, K. Danzaki, M. Inoue, E. D. Cardakli, T. Nonaka, N. Aggarwal, W. E. Barclay, R. R. Ji, M. L. Shinohara

This study shows Dectin-1 protects against CNS autoimmunity through a non-canonical pathway independent of Card9, promoting neuroprotective molecules like Osm in myeloid cells and reducing EAE severity via astrocytic OsmR signaling.

bioRxiv · May 6, 2020 · PreprintImmunology