Author

Thomas Ruelicke

University of Veterinary Medicine Vienna

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:clustering· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Ana VillalbaiD· 2 shared Carmen Streicher· 2 shared Florian M. PauleriD· 2 shared Osvaldo A. MirandaiD· 2 shared Simon HippenmeyeriD· 2 shared Amarbayasgalan DavaatsereniD· 1 shared Anna HegeriD· 1 shared Bassem A. HassaniD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersNeuroscience

Ana VillalbaORCID

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2 papers

Recent·Most cited

Pten Orchestrates Neurogenic Radial Glia Lineage Progression and Tunes Neocortical Astrocyte Production

Osvaldo A. Miranda, Ximena Contreras, Florian M. Pauler, Amarbayasgalan Davaatseren, Nicole Amberg, Carmen Streicher, Ana Villalba, Anna Heger, Corentine Marie, Bassem A. Hassan, Thomas Ruelicke, Simon Hippenmeyer

This study investigates the role of Pten in radial glial progenitor lineage progression and astrocyte production during cortical development, finding that Pten loss increases astrocyte generation and disrupts growth factor signaling.

bioRxiv · May 5, 2026 · PreprintNeuroscience

Mtor/Rptor Function Globally Prevents Cortical Microcephaly and Cell-autonomously Promotes Postnatal Neuron Survival in Cell Type Specific Manner

Ana Villalba, Robert Beattie, Florian M. Pauler, Carmen Streicher, Osvaldo A. Miranda, Thomas Krausgruber, Martin Senekowitsch, Matthias Farlik, Christoph Bock, Thomas Ruelicke, Simon Hippenmeyer

This study investigates the role of Mtor/Rptor in cortical development, finding that its loss causes microcephaly through tissue-wide effects, while cell-autonomous Rptor function is critical for postnatal neuron survival in a cell-type-specific manner.

bioRxiv · May 5, 2026 · PreprintNeuroscience