Author

Ulrike Beutling

Helmholtz Centre for Infection Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Mark BrönstrupiD· 2 shared Raimo FrankeiD· 2 shared André FleißneriD· 1 shared Birte Dowerg· 1 shared Connor S.R. JankowskiiD· 1 shared Eva MedinaiD· 1 shared Fangfang CheniD· 1 shared Hanna F Willenbockel· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCell Biology

Mark BrönstrupORCID

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2 papers

Recent·Most cited

Itaconate is metabolized to 2-hydroxymethylsuccinate through a CoA-independent degradation pathway in mitochondria

Fangfang Chen, Laura Hadam, Birte Dowerg, Vadim S. Korotkov, Connor S.R. Jankowski, Ulrike Beutling, Hanna F Willenbockel, Ulrike Brandt, Eva Medina, André Fleißner, Henrikus Garritsen, Michael Jarek, Raimo Franke, Thomas Weichhart, Mark Brönstrup, , Thekla Cordes

The paper identifies 2-hydroxymethylsuccinate as a product of itaconate metabolism via a CoA-independent pathway and shows tissue-specific degradation routes.

bioRxiv · Sep 29, 2025 · PreprintCell Biology

Metabolic reprogramming of methylthioadenosine-dependent sulfur recycling is a major driver of CHIKV infection

Jackson M. Muema, Myriam Friemelt, Heike Overwin, Ulrike Beutling, Raimo Franke, Mark Brönstrup, Peter Sandner, Ursula Bilitewski

This study identifies metabolic reprogramming of sulfur recycling via MTA and Mat2a as a key driver of CHIKV infection and highlights potential therapeutic targets.

bioRxiv · Jul 11, 2025 · PreprintMicrobiology