Author

X.-R. Peng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Anisimov· 1 shared A. Oldner· 1 shared A. Osman· 1 shared A. Park· 1 shared A.-C. Andreasson· 1 shared B. Guigas· 1 shared B. Spoorenberg· 1 shared C. Betsholtz· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPathology

A. Anisimov

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

FGF21 protects against hepatic lipotoxicity and macrophage activation to attenuate fibrogenesis in nonalcoholic steatohepatitis

C. Liu, M. Schönke, B. Spoorenberg, J. Lambooij, H. van der Zande, E. Zhou, M. E. Tushuizen, A.-C. Andreasson, A. Park, S. Oldham, M. Uhrbom, I. Ahlstedt, Y. Ikeda, K. Wallenius, X.-R. Peng, , P. C. Rensen

This study shows that FGF21 overexpression in the liver reduces lipotoxicity, inflammation, and fibrosis in NASH by blocking lipid influx and macrophage activation.

bioRxiv · Sep 20, 2022Pathology

AdipoR2 is Essential for Membrane Lipid Homeostasis in Response to Dietary Saturated Fats

R. Devkota, M. Ruiz, H. Palmgren, M. Stahlman, M. Maresca, H. Jaiswal, M. Bohlooly-Y, X.-R. Peng, J. Boren, M. Pilon

The paper identifies AdipoR2 as essential for maintaining membrane lipid homeostasis by counteracting saturated fat-induced membrane rigidification across species.

bioRxiv · Jun 11, 2020Cell Biology

Pericyte-specific vascular expression of SARS-CoV-2 receptor ACE2 - implications for microvascular inflammation and hypercoagulopathy in COVID-19

L. He, M. A. Mae, L. Muhl, Y. Sun, R. Pietila, K. Nahar, E. V. Liebanas, M. J. Fagerlund, A. Oldner, J. Liu, G. Genove, L. Zhang, Y. Xie, S. Liptidis, G. Mocci, , C. Betsholtz

The paper shows that pericytes, not endothelial cells, express ACE2 in microvessels, suggesting they may be infected in COVID-19 and contribute to microvascular inflammation and hypercoagulopathy.

bioRxiv · May 11, 2020 · PreprintPathology