Author

Y. Chaudhry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

E. Emmott· 2 shared I. G. Goodfellow· 2 shared M. Hosmillo· 2 shared A. Merenda· 1 shared A. W. Baird· 1 shared B.-K. Koo· 1 shared C. B. Willen· 1 shared C. S. McKimmie· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersImmunology

E. Emmott

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A novel antiviral formulation inhibits a range of enveloped viruses.

N. F. Fletcher, L. W. Meredith, E. L. Tidswell, S. R. Bryden, D. Goncalves-Carneiro, Y. Chaudhry, C. Shannon-Lowe, M. A. Folan, D. A. Lefteri, M. Pingen, D. Bailey, C. S. McKimmie, A. W. Baird

This study evaluates a novel antiviral formulation, ViroSAL, which inhibits multiple enveloped viruses in vitro and in vivo by disrupting viral envelopes.

bioRxiv · Mar 29, 2020Microbiology

Norovirus replication in human intestinal epithelial cells is restricted by the interferon-induced JAK/STAT signalling pathway and RNA Polymerase II mediated transcriptional responses.

M. T. Hosmillo, Y. Chaudhry, K. Nayak, F. Sorgeloos, B.-K. Koo, A. Merenda, R. Lillestol, L. Drumright, M. Zilbauer, I. G. Goodfellow

This study shows that HuNoV replication in intestinal epithelial cells is restricted by IFN-induced transcriptional responses and that inhibiting these responses enhances viral replication.

bioRxiv · Aug 10, 2019 · PreprintMicrobiology

Ifit1 regulates norovirus infection and enhances the interferon response in murine macrophage-like cells.

H. V. Mears, E. Emmott, Y. Chaudhry, M. Hosmillo, I. G. Goodfellow, T. R. Sweeney

This study shows Ifit1 enhances the interferon response but does not directly inhibit norovirus translation in murine macrophage-like cells.

bioRxiv · Apr 17, 2019Immunology

Noroviruses subvert the core stress granule component G3BP1 to promote viral VPg-dependent translation.

M. Hosmillo, J. Lu, M. R. McAllaster, J. B. Eaglesham, X. Wang, E. Emmott, P. Domingues, Y. Chaudhry, T. J. Fitzmaurice, M. K. H. Tung, M. Panas, G. McInerney, N. Locker, C. B. Willen, I. Goodfellow

This study identifies G3BP1 as a host factor essential for norovirus infection and reveals its role in VPg-dependent translation initiation.

bioRxiv · Mar 8, 2019Microbiology