Author

Y. Iwatani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1 Physiology· 1 Zoology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:C. elegans· 2 Research Focus:development & morphogenesis· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. S. Nishino· 2 shared H. Ode· 1 shared K. Fujimoto· 1 shared K. Fukuda· 1 shared K. Imai· 1 shared K. Matsushita· 1 shared K. Miyoshi· 1 shared K. Tokunaga· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersPhysiology

A. S. Nishino

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pulsation waves along the Ciona heart tube reverse by bimodal rhythms expressed by a remote pair of pacemakers

Y. Fujikake, K. Fukuda, K. Matsushita, Y. Iwatani, K. Fujimoto, A. S. Nishino

The paper investigates how pulsation waves in the Ciona heart tube reverse due to bimodal rhythms from terminal pacemakers.

bioRxiv · Oct 6, 2023 · PreprintPhysiology

An autonomous tail-beating cycle period expressed by a region-specific swimming pattern generator in the Ciona larva

T. Hara, S. Hasegawa, Y. Iwatani, A. S. Nishino

The paper identifies a region-specific swimming pattern generator in Ciona larva mid-piece fragments that autonomously produce periodic tail beating without external stimuli.

bioRxiv · Apr 12, 2021 · PreprintZoology

Naturally mutated spike proteins of SARS-CoV-2 variants show differential levels of cell entry

S. Ozono, Y. Zhang, H. Ode, T. S. Tan, K. Imai, K. Miyoshi, S. Kishigami, T. Ueno, Y. Iwatani, T. Suzuki, K. Tokunaga

The paper examines how naturally occurring mutations in SARS-CoV-2 spike proteins affect cell entry via ACE2 and TMPRSS2, finding that the D614G mutation increases infectivity but remains neutralization-sensitive.

Nature Communications · Feb 8, 2021Peer-reviewedMicrobiology