Author

Y. Koyanagi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Organism:chicken / avian· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

K. Sato· 3 shared I. Kimura· 2 shared J. Ito· 2 shared A. Coudray· 1 shared A. Soper· 1 shared D. Trono· 1 shared H. Aso· 1 shared H. Nakaoka· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersMicrobiology

K. Sato

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single-cell transcriptome analysis illuminating the characteristics of species-specific innate immune responses against viral infections

H. Aso, J. Ito, H. Ozaki, Y. Kashima, Y. Suzuki, Y. Koyanagi, K. Sato

This study compares immune responses between primates and bats using single-cell RNA sequencing, revealing differences in antiviral gene induction and monocyte subsets.

bioRxiv · Dec 6, 2022Microbiology

SARS-CoV-2 ORF3b is a potent interferon antagonist whose activity is further increased by a naturally occurring elongation variant

Y. Konno, I. Kimura, K. Uriu, M. Fukushi, T. Irie, Y. Koyanagi, S. Nakagawa, K. Sato

This paper shows that SARS-CoV-2 ORF3b is a potent interferon antagonist, with a naturally occurring longer variant enhancing its anti-interferon activity in severe COVID-19 cases.

bioRxiv · May 11, 2020Microbiology

Endogenous retroviruses drive KRAB zinc-finger family protein expression for tumor suppression

J. Ito, I. Kimura, A. Soper, A. Coudray, Y. Koyanagi, H. Nakaoka, I. Inoue, P. Turelli, D. Trono, K. Sato

The paper shows HERV activation in tumors upregulates KZFPs, which suppress cancer progression by downregulating cell cycle and adhesion genes, improving disease outcomes.

bioRxiv · Feb 2, 2020Cancer Biology