Author

Y. Minato

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:drug response / drug discovery· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

A. D. Baughn· 3 shared J. M. Thiede· 2 shared M. D. Howe· 2 shared A. A. Bauman· 1 shared A. Baughn· 1 shared A. Nishiyama· 1 shared C. C. Aldrich· 1 shared D. M. Gohl· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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3 shared papersMicrobiology

A. D. Baughn

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4 papers

Recent·Most cited

Functional genomics reveals strain-specific genetic requirements conferring hypoxic growth in Mycobacterium intracellulare

Y. Tateishi/Yoshitaka, Y. Ozeki, A. Nishiyama, Y. Morishige, Y. Minato, A. Baughn, S. Matsumoto

The paper identifies strain-specific genetic requirements for hypoxic growth in Mycobacterium intracellulare using transposon sequencing and validates findings in a mouse infection model.

bioRxiv · May 12, 2024Microbiology

Pyrazinamide action is driven by the cell envelope stress response in Mycobacterium tuberculosis

J. M. Thiede, N. Dillon, M. D. Howe, S. J. Modlin, R. Aflakpui, S. Hoffner, F. Valafar, Y. Minato, A. D. Baughn

This study identifies that the cell envelope stress response drives pyrazinamide susceptibility in Mycobacterium tuberculosis, independent of pH, and shows that peptidoglycan synthesis inhibitors enhance its action.

bioRxiv · Feb 17, 2021 · PreprintMicrobiology

Genome-wide assessment of Mycobacterium tuberculosis conditionally essential metabolic pathways

Y. Minato, D. M. Gohl, J. M. Thiede, J. M. Chacon, W. R. Harcombe, F. Maruyama, A. D. Baughn

This study identifies conditionally essential metabolic pathways in Mycobacterium tuberculosis using Tn-seq and in silico models, revealing gaps in current knowledge of its metabolic capabilities.

bioRxiv · May 14, 2019 · PreprintMicrobiology

Methionine antagonizes para-aminosalicylic acid activity via affecting folate precursor biosynthesis pathway in Mycobacterium tuberculosis

M. D. Howe, S. L. Kordus, M. S. Cole, A. A. Bauman, C. C. Aldrich, A. D. Baughn, Y. Minato

This study identifies methionine's role in antagonizing PAS activity by affecting folate precursor biosynthesis in M. tuberculosis.

bioRxiv · May 17, 2018Microbiology