Author

Zihan Xu

Kobe University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Adam Ramadan· 1 shared Anghesom GhebremedhiniD· 1 shared Binben Wang· 1 shared Birkley S. LimiD· 1 shared Changle Ke· 1 shared Chieko MatsuiiD· 1 shared Danyang XiongiD· 1 shared Dewa Nyoman Murti AdyaksaiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCancer Biology

Adam Ramadan

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Acetylation of NDUFA13 at K7 Senses OXPHOS Dysfunction to Disrupt Prohibitin Scaffolds and Drive Cristae Remodelling in Heart Failure

Jinliang Nan, Xinnuo Yang, Yinuo Lin, Jian Shen, Hongpeng Chen, Zihan Xu, Jian Ye, Yaping Wang, Danyang Xiong, Changle Ke, Lianlian Zhu, Xiangmin Kong, Zhiwei Zhong, Tanchen Ren, Jun Jiang, , Xiao He

This study identifies NDUFA13-K7 acetylation as a sensor linking OXPHOS dysfunction to cristae remodelling and metabolic changes in heart failure.

bioRxiv · Oct 28, 2026 · PreprintCell Biology

An IL-34-IGF-1 inflammatory axis fuels KRAS-mutant lung cancer progression

Jaroslav Žák, Hui Chen, Erpei Wang, Patrick Ozark, Giuliana P. Mognol, MATTHEW Dae-Young PARK, Nadine Fournier, Priyanka Chaudary, Jingjing Hu, Ryan M. Shepard, Anghesom Ghebremedhin, Marc A. Paradise, Jason Rivera, William J Harris, Zihan Xu, , Judith A. Varner

This study identifies an IL-34-IGF-1 axis between macrophages and KRAS-mutant lung tumor cells that promotes tumor progression and poor survival.

bioRxiv · Jul 15, 2026 · PreprintCancer Biology

HCV infection induces ubiquitin-dependent degradation of LATS1, inactivating the Hippo pathway and upregulating transcription of the CYR61 and CTGF genes

Maria Alethea Septianastiti, Chieko Matsui, Zihan Xu, Fransisca Puspitasari, Dewa Nyoman Murti Adyaksa, Lin Deng, Takayuki Abe, Ikuo Shoji

This study shows HCV infection causes LATS1 degradation via ubiquitin-mediated proteasomal pathway, inactivating the Hippo pathway and increasing CYR61 and CTGF gene transcription.

Journal of General Virology · Feb 5, 2026Peer-reviewedMicrobiology