Author

Moritz F. Schlapansky

ETH ZurichORCID 0000-0001-6072-7840Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Jacob E. CorniD· 2 shared Simon C.C. LamiD· 2 shared Stephen P. JacksoniD· 2 shared Aitana Egea· 1 shared Aldo S. BaderiD· 1 shared Alexandra Hart· 1 shared Ana GvozdenoviciD· 1 shared Andreas VillungeriD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenomics

Jacob E. CornORCID

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2 papers

Recent·Most cited

GenomicsbioRxivPreprintOct 23, 2025

Cell-stereotyped DNA repair outcomes are widespread during genome editing

Moritz F. Schlapansky, Markus Schröder, António J. Santinha…·Jacob E. Corn

The paper develops scOUT-seq to profile DNA repair outcomes in single cells and finds that repair outcomes vary significantly across cell types and subtypes.

1 citation

humanmouse

𝕏 PostBluesky

Cancer BiologyNature CommunicationsPeer-reviewedOct 9, 2025

WEE1 inhibitors synergise with mRNA translation defects via activation of the kinase GCN2

Jordan Wilson, Jiayi Zhu, Vanesa Vinciauskaite…·Stephen P. Jackson

This study shows WEE1 inhibitors work better with mRNA translation defects by activating GCN2, and suggests ways to reduce off-target effects.

3 citations

humanCRISPR screen

𝕏 PostBluesky